94-20449. Drug Products for the Treatment and/or Prevention of Nocturnal Leg Muscle Cramps for Over-the-Counter Human Use; Final Rule DEPARTMENT OF HEALTH AND HUMAN SERVICES  

  • [Federal Register Volume 59, Number 161 (Monday, August 22, 1994)]
    [Unknown Section]
    [Page 0]
    From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
    [FR Doc No: 94-20449]
    
    
    [[Page Unknown]]
    
    [Federal Register: August 22, 1994]
    
    
    _______________________________________________________________________
    
    Part IV
    
    
    
    
    
    Department of Health and Human Services
    
    
    
    
    
    _______________________________________________________________________
    
    
    
    Food and Drug Administration
    
    
    
    _______________________________________________________________________
    
    
    
    21 CFR Part 310
    
    
    
    
    Drug Products for the Treatment and/or Prevention of Nocturnal Leg 
    Muscle Cramps for Over-the-Counter Human Use; Final Rule
    DEPARTMENT OF HEALTH AND HUMAN SERVICES
    
    Food and Drug Administration
    
    21 CFR part 310
    
    [Docket No. 77N-0094]
    RIN 0905-AA06
    
     
    Drug Products for the Treatment and/or Prevention of Nocturnal 
    Leg Muscle Cramps for Over-The-Counter Human Use
    
    AGENCY: Food and Drug Administration, HHS.
    
    ACTION: Final rule.
    
    -----------------------------------------------------------------------
    
    SUMMARY:  The Food and Drug Administration (FDA) is issuing a final 
    rule establishing that any over-the-counter (OTC) drug product for the 
    treatment and/or prevention of nocturnal leg muscle cramps is not 
    generally recognized as safe and effective and is misbranded. FDA is 
    issuing this final rule after considering public comments on the 
    agency's proposed regulation, which was issued in the form of a 
    tentative final monograph, and all new data and information on drug 
    products for the treatment and/or prevention of nocturnal leg muscle 
    cramps that have come to the agency's attention. This final rule is 
    part of the ongoing review of OTC drug products conducted by FDA.
    
    EFFECTIVE DATE: February 22, 1995.
    FOR FURTHER INFORMATION CONTACT: William E. Gilbertson, Center for Drug 
    Evaluation and Research (HFD-810), Food and Drug Administration, 5600 
    Fishers Lane, Rockville, MD 20857, 301-594-5000.
    
    SUPPLEMENTARY INFORMATION: In the Federal Register of October 1, 1982 
    (47 FR 43562), FDA published, under Sec. 330.10(a)(6) (21 CFR 
    330.10(a)(6)), an advance notice of proposed rulemaking to reopen the 
    rulemaking for OTC internal analgesic, antipyretic, and antirheumatic 
    drug products to consider the OTC use of quinine for the treatment of 
    nocturnal leg muscle cramps, together with the recommendations of the 
    Advisory Review Panel on OTC Miscellaneous Internal Drug Products 
    (Miscellaneous Internal Panel), which was the advisory review panel 
    responsible for evaluating data on the active ingredients in this drug 
    class. Interested persons were invited to submit comments by December 
    30, 1982. Reply comments in response to comments filed in the initial 
    comment period could be submitted by January 31, 1983.
        In accordance with Sec. 330.10(a)(10), the data and information 
    considered by the Panel were put on display in the Dockets Management 
    Branch (HFA-305), Food and Drug Administration, rm. 1-23, 12420 
    Parklawn Dr., Rockville, MD 20857, after deletion of a small amount of 
    trade secret information.
        The agency's proposed regulation, in the form of a tentative final 
    monograph, for OTC drug products for the treatment and/or prevention of 
    nocturnal leg muscle cramps was published in the Federal Register of 
    November 8, 1985 (50 FR 46588). Interested persons were invited to file 
    by January 7, 1986, written comments, objections, or requests for oral 
    hearing before the Commissioner of Food and Drugs regarding the 
    proposal. Interested persons were invited to file comments on the 
    agency's economic impact determination by March 10, 1986. New data 
    could have been submitted until November 10, 1986, and comments on the 
    new data could have been submitted until January 8, 1987. Final agency 
    action occurs with the publication of this final rule on OTC drug 
    products for the treatment and/or prevention of nocturnal leg muscle 
    cramps.
        In the preamble to the agency's proposed rule on OTC drug products 
    for the treatment and/or prevention of nocturnal leg muscle cramps (50 
    FR 46588), the agency stated that no active ingredient in drug products 
    used OTC for the treatment and/or prevention of nocturnal leg muscle 
    cramps had been found to be generally recognized as safe and effective 
    and not misbranded, but that Category I labeling was being proposed in 
    that document in the event that data were submitted that resulted in 
    the upgrading of any ingredients to monograph status in the final rule. 
    In this final rule, no ingredient in OTC drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps has been 
    determined to be generally recognized as safe and effective. Therefore, 
    proposed part 343 (21 CFR 343), subpart E for OTC drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps is not being 
    issued as a final regulation.
        This final rule declares OTC drug products containing active 
    ingredients for the treatment and/or prevention of nocturnal leg muscle 
    cramps to be new drugs under section 201(p) of the Federal Food, Drug, 
    and Cosmetic Act (the act) (21 U.S.C. 321(p)), for which an application 
    or abbreviated application (hereinafter called application) approved 
    under section 505 of the act (21 U.S.C. 355) and 21 CFR part 314 is 
    required for marketing. In the absence of an approved application, 
    products containing drugs for this use also would be misbranded under 
    section 502 of the act (21 U.S.C. 352). In appropriate circumstances, a 
    citizen petition to establish a monograph may be submitted under 21 CFR 
    10.30 in lieu of an application.
        This final rule amends 21 CFR part 310 to include drug products 
    containing active ingredients for the treatment and/or prevention of 
    nocturnal leg muscle cramps by adding new Sec. 310.546 (21 CFR 310.546) 
    to subpart E. The inclusion of OTC drug products for the treatment and/
    or prevention of nocturnal leg muscle cramps in part 310 is consistent 
    with FDA's established policy for regulations in which there are no 
    monograph conditions. (See, e.g., Secs. 310.510, 310.519, 310.525, 
    310.526, 310.532, 310.533, and 310.534.) If, in the future, any 
    ingredient is determined to be generally recognized as safe and 
    effective for use in an OTC drug product for the treatment and/or 
    prevention of nocturnal leg muscle cramps, the agency will promulgate 
    an appropriate regulation at that time.
        The OTC drug procedural regulations (21 CFR 330.10) provide that 
    any testing necessary to resolve the safety or effectiveness issues 
    that formerly resulted in a Category III classification, and submission 
    to FDA of the results of that testing or any other data, must be done 
    during the OTC drug rulemaking process before the establishment of a 
    final monograph. Accordingly, FDA does not use the terms ``Category I'' 
    (generally recognized as safe and effective and not misbranded), 
    ``Category II'' (not generally recognized as safe and effective or 
    misbranded), and ``Category III'' (available data are insufficient to 
    classify as safe and effective, and further testing is required) at the 
    final monograph stage. In place of Category I, the term ``monograph 
    conditions'' is used; in place of Categories II or III, the term 
    ``nonmonograph conditions'' is used.
        In the proposed rule for OTC drug products for the treatment and/or 
    prevention of nocturnal leg muscle cramps (50 FR 46588), the agency 
    advised that it would provide a period of 12 months after the date of 
    publication of the final monograph in the Federal Register for 
    relabeling and reformulation of drug products for the treatment and/or 
    prevention of nocturnal leg muscle cramps to be in compliance with the 
    monograph. Although several manufacturers submitted data and 
    information in response to the proposed rule, the data and information 
    were not sufficient to support monograph conditions, and no monograph 
    is being established at this time. Therefore, drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps that are 
    subject to this rule are not generally recognized as safe and effective 
    and are misbranded (nonmonograph conditions). The agency also stated 
    that if a safety problem is identified for a particular nonmonograph 
    condition, a shorter deadline may be set for removal of that condition 
    from OTC drug products. As stated below, a safety problem has been 
    identified for OTC drug products containing quinine sulfate for the 
    treatment and/or prevention of nocturnal leg muscle cramps. Therefore, 
    the agency has determined that initial introduction or initial delivery 
    for introduction into interstate commerce of quinine sulfate for the 
    treatment and/or prevention of nocturnal leg muscle cramps must cease 
    effective February 22, 1995. After that date, no OTC drug products that 
    are subject to this final rule may be initially introduced or initially 
    delivered for introduction into interstate commerce unless they are the 
    subject of an approved application. The agency is unaware of any 
    quinine sulfate drug product for this indication that is the subject of 
    an approved application. Any such drug product in interstate commerce 
    after the effective date of this final rule that is not in compliance 
    with the regulation is subject to regulatory action.
        In response to the proposed rule on OTC drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps, three drug 
    manufacturers and a nutrition information service submitted comments. 
    One comment included a request for an oral hearing before the 
    Commissioner of Food and Drugs.
        After the administrative record closed, a citizen petition was 
    submitted on December 1, 1988. In response to this petition, nine 
    additional comments were submitted. The Commissioner found that the 
    petition and subsequent comments raised safety and effectiveness issues 
    that warranted consideration before the final rule issued. Accordingly, 
    under Sec. 330.10(a)(7)(v), the Commissioner determined that good cause 
    was shown to warrant consideration of the petition and the additional 
    comments before the final rule issued. Copies of the comments received 
    and the petition are on public display in the Dockets Management Branch 
    (address above). Additional information that has come to the agency's 
    attention since publication of the proposed rule is also on public 
    display in the Dockets Management Branch.
    
    I. The Agency's Conclusions on the Comments
    
    A. General Comments
    
        1. One comment disagreed with the agency's determination that 
    adequate clinical data did not exist to support the Category I status 
    of quinine for both safety and effectiveness for OTC use in the 
    treatment and/or prevention of nocturnal leg muscle cramps (50 FR 46588 
    at 46590). The comment expressed the belief that sufficient evidence 
    already exists for this use of quinine. In support of its position, the 
    comment referred to information it had submitted on December 27, 1982, 
    in response to the advance notice of proposed rulemaking for this class 
    of drug products.
        The agency discussed this information in the tentative final 
    monograph (50 FR 46588 at 46589) and concluded that it did not provide 
    sufficient evidence to establish that quinine is generally recognized 
    as safe and effective for OTC use for the treatment and/or prevention 
    of nocturnal leg muscle cramps. The agency identified the issues to be 
    addressed in studies before quinine could be reclassified from Category 
    III to Category I (50 FR 46590 and 46591). The comment did not provide 
    any new information to address these issues. New data and information 
    submitted by other interested persons are discussed in section I.B., 
    comments 4 through 9 of this document.
    
    B. Comments on the Safety of Nocturnal Leg Muscle Cramp Ingredients
    
        2. Two comments contended that FDA accepted the safe OTC use of 
    quinine sulfate for nocturnal leg cramps when it published, and did not 
    dissent from, the Miscellaneous Internal Panel's conclusions and 
    recommendations that ``* * * quinine appears to be reasonably safe over 
    prolonged periods of time in generally recommended doses of 200 to 325 
    mg daily'' (47 FR 43562 at 43564).
        Contrary to the comments' contention, the record makes it clear 
    that neither the agency nor the advisory panels accepted the safety of 
    OTC use of quinine for nocturnal leg muscle cramps. The July 8, 1977 
    report of the Advisory Review Panel on OTC Internal Analgesic and 
    Antirheumatic Drug Products (42 FR 35346 at 35434) summarized the 
    safety of quinine and stated ``Although quinine has demonstrated 
    analgesic, antipyretic and muscle relaxant actions, its numerous toxic 
    effects give it an unfavorable benefit to risk ratio for these 
    purposes.'' The Panel concluded that ``Until controlled studies show 
    that a dose of not more than 325 mg daily is safe and useful for relief 
    of nocturnal leg cramps the drug should not be available for OTC use 
    for treatment of nocturnal leg cramps.'' While the Miscellaneous 
    Internal Panel's report stated that quinine ``* * * appears to be 
    reasonably safe * * *'' (47 FR 43564), the Panel did not conclude that 
    quinine was safe in doses used for the treatment of nocturnal leg 
    muscle cramps. The Panel's recommendation that quinine should be placed 
    in Category III for use in the treatment of nocturnal leg muscle cramps 
    cited the need for more information about both safety and efficacy in 
    its concluding statement (47 FR 43564).
        In the tentative final monograph for OTC drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps, the agency 
    concurred with: (1) The Miscellaneous Internal Panel's classification 
    of quinine sulfate as Category III, and (2) the Internal Analgesic 
    Panel's conclusion that the drug should not be generally recognized as 
    safe and effective (Category II) for this use until its safety and 
    efficacy are demonstrated in controlled clinical trials (50 FR 46588 at 
    46592). It is clear, therefore, that neither the agency nor its 
    advisory panels have determined that quinine may be safely used for 
    this indication.
        3. Three comments stated that quinine has been safely used by 
    millions of consumers for a variety of conditions, including leg 
    cramps, for more than 50 years, and that this long history of usage 
    demonstrates the safety of quinine.
        General reference to the history of use of quinine cannot be 
    accepted as evidence of its safety. Historically, there has been no 
    clear location for the organized collection and analysis of adverse 
    drug experience reports on OTC drug products. Adverse events associated 
    with OTC drug products are still vastly underreported for a number of 
    reasons. First, for most OTC drugs there is no requirement that 
    manufacturers and distributors report adverse events to the FDA, even 
    those that are serious or life threatening. Second, physicians, who are 
    the principal reporters to the United States spontaneous reporting 
    system, may not become aware of reactions to OTC drugs. Patients often 
    do not mention OTC drugs in giving medication histories. If they do, it 
    is often not clear to physicians to which manufacturer the adverse 
    event should be reported.
        4. Two comments in support of keeping quinine available OTC 
    discussed the adverse event reports on file for quinine in FDA's 
    spontaneous reporting system. Lavy (Ref. 1) stated that there were 52 
    reports suggestive of hypersensitivity reactions, including 8 deaths, 
    among the reports on file from 1969 to 1988. Lavy concentrated on 
    reports of thrombocytopenia (a decrease in the number of blood 
    platelets) and stated that bleeding secondary to thrombocytopenia may 
    have been related to four of the eight deaths reported in this 20-year 
    period. The comment stated that only one case provided sufficient 
    information to fulfill all diagnostic criteria for drug-induced 
    immunologic thrombocytopenia (consistent clinical history, exclusion of 
    other causes, positive in vitro test and, theoretically, the 
    demonstration of recurrent thrombocytopenia after rechallenge). Lavy 
    pointed out that adverse event reports should reflect a significant 
    prevalence of severe quinine-associated purpura (if occurring) because 
    this reaction is readily identifiable by the patient, physician, and 
    hospital. Using industry quinine sales figures and data from the FDA 
    spontaneous reporting system, Lavy concluded that the number of 
    reported quinine-related hypersensitivity reactions is quite low, even 
    if greatly underreported.
        Aster (Ref. 2) reviewed adverse drug reactions reported to FDA from 
    1969 through December 1989 and estimated that approximately 1,000 
    reactions in 313 individuals were reported for quinine. Aster's 
    estimate was not limited to hypersensitivity reactions, but included 
    all reported reactions (including multiple reactions per individual). 
    Aster did not report the specific search criteria used to obtain the 
    adverse drug reaction reports other than to state that the ``FDA 
    materials provide a cumulative listing of all adverse drug reactions 
    (ADR) reported in connection with quinine since 1969.'' After 
    eliminating reactions considered highly unlikely to bear a cause-and-
    effect relationship to quinine, Aster identified 254 reactions that he 
    considered ``significant.'' There were 83 hematologic reactions and 46 
    additional reactions possibly associated with hypersensitivity. Of 63 
    possible cases of thrombocytopenia, 36 included information verifying 
    low platelet levels. Aster identified 50 fatalities (15 from 
    hematologic complications, 3 from hypersensitivity, and 32 from other 
    causes). Overall, Aster classified 51 percent of the adverse reactions 
    as idiosyncratic and eight percent as the result of overdose. The 
    remaining 41 percent were of indeterminate etiology and consisted of 
    liver and kidney dysfunction, neurologic disorders, and various 
    hemorrhagic manifestations. Of the reports in which inadequate 
    information was provided for full evaluation, Aster noted the 
    possibility that some of the six cerebrovascular accidents reported may 
    have been associated with thrombocytopenia induced by quinine. Aster 
    concluded that: (1) No information is available that would enable the 
    identification of people at risk to sensitivity reactions to quinine; 
    (2) the rarity of sensitivity reactions and the rapidity with which 
    they occur make early detection of such reactions impossible and, (3) 
    the dramatic symptomatology of such reactions, when they occur, leads 
    people to seek prompt medical attention. Aster concluded, therefore, 
    that serious adverse reactions would neither be prevented nor reduced 
    in incidence by restricting quinine availability to prescription 
    status.
        The agency has reviewed the comments and the reports of adverse 
    reactions to quinine products (listed in the agency's spontaneous 
    reporting system under quinine, quinine sulfate, and three brand name 
    products used for the treatment and/or prevention of nocturnal leg 
    muscle cramps). From 1969 through June 1992, FDA received 157 adverse 
    reaction reports in which quinine was listed as a suspect drug. There 
    were 84 serious reactions: 23 deaths, 5 cases in which the person was 
    disabled, and 56 hospitalizations not involving death or disablement. 
    Of the 157 adverse reaction reports, 52 (approximately 33 percent) did 
    not contain dosage and/or product name information, or reported daily 
    dosages in excess of those typically recommended for the treatment and/
    or prevention of nocturnal leg muscle cramps. The remaining 105 reports 
    listed the names of quinine products labeled for use in the treatment 
    and/or prevention of nocturnal leg muscle cramps and/or daily dosages 
    recommended by these products, and included 60 serious reactions 
    involving 16 deaths, 4 cases of disablement, and 40 hospitalizations 
    not involving death or disablement. More importantly, 56 of the 105 
    reports (approximately 53 percent) have been received since 1988, and 
    there is an alarming trend of increasing numbers of reports per year 
    since 1986 as the market for OTC drug products containing quinine for 
    the treatment and/or prevention of nocturnal leg muscle cramps has 
    expanded during that period. Approximately 70 percent (42 of 60) of all 
    reports of serious reactions, 44 percent (7 of 16) of all reported 
    deaths, and 78 percent (31 of 40) of all reported hospitalizations on 
    file since 1969 were reported in the 4 1/2-year period between January 
    1988 and July 1992. There were 20 cases (19 percent of reports on file) 
    reported in 1991 alone: 3 were disabling, 11 required hospitalization, 
    and 1 resulted in death.
        Nocturnal leg muscle cramps are a common condition in the elderly 
    (Ref. 3). Presumably, with an increasing average age in the American 
    population, the market for OTC drug products containing quinine for the 
    treatment and/or prevention of nocturnal leg muscle cramps also 
    increased during this period. The number of adverse reaction reports 
    for people 60 years of age or older, involving quinine products and/or 
    quinine dosages used in the treatment and/or prevention of nocturnal 
    leg muscle cramps, has increased by a factor of five (from 2 to 10) 
    during the period between January 1988 and December 1991.
        The agency conducted a detailed review of 110 reports on file from 
    1969 through 1990; 69 (approximately 63 percent) of these reports 
    involved hypersensitivity reactions ranging from rash and fever to 
    angioneurotic edema, thrombocytopenia, or generalized anaphylaxis. Of 
    these 69 reports, 57 (approximately 83 percent) involved quinine 
    products and/or quinine dosages used in the treatment and/or prevention 
    of nocturnal leg muscle cramps. An attempt was made to identify only 
    those reports in which the relationship between quinine and the 
    reported event was strong and reasonably unrelated to other factors. 
    Factors considered included the temporal relationship between quinine 
    administration and the event, absence of concomitant medications (or 
    abatement of the adverse event after quinine was discontinued), absence 
    of confounding medical conditions, a positive test for quinine mediated 
    antibodies, or history of a similar reaction associated with previous 
    quinine exposure. Using these factors, of the 110 reports 26 were 
    identified in which it can be reasonably concluded that quinine was the 
    causative agent. These included 6 moderately severe to severe skin 
    reactions, 2 of which were erythema multiforme-like reactions; 13 
    hematologic events, with 2 resulting in death; 2 cases of hepatitis or 
    elevated liver enzymes; 2 renal reactions, one leading to renal failure 
    requiring dialysis, the other leading to death; 2 cases of a 
    hypersensitivity syndrome with symptoms that included chills, nausea, 
    vomiting, and diarrhea; and 1 report of anaphylaxis complicated by 
    seizures and hypoxia following a single dose of quinine. None of these 
    cases reported an overdose of the drug, and 21 of the 26 reports 
    (approximately 81 percent) involved quinine products and/or quinine 
    dosages used in the treatment and/or prevention of nocturnal leg muscle 
    cramps.
        Even using strict criteria to identify cases in which a causal 
    relationship of quinine to adverse event is likely, FDA finds that 
    quinine is associated with serious adverse events. There is no 
    compelling reason to restrict evaluation of the safety of quinine to 
    reported cases of thrombocytopenia, as Lavy did. The other adverse 
    effects are also serious and must be considered in weighing the 
    benefits and risks of products containing quinine. The agency agrees 
    with Aster that there is currently no way in advance to identify people 
    at risk of hypersensitivity reactions and, therefore, no effective way 
    to warn against use by such individuals (see section I.B., comment 10). 
    It does not agree, however, that physician monitoring might not 
    minimize serious reactions. Thrombocytopenia, for example, can lead to 
    bruising and other evidence of cutaneous bleeding. A physician could 
    warn patients to report such signs and stop the drug, perhaps 
    preventing a significant hemorrhagic event.
    
    References
    
        (1) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treatment and/or Prevention of Nocturnal Leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (2) Aster, R. H., ``Q-Vel: Could Serious Adverse Reactions be 
    Prevented by Having the Drug Available Only on Prescription,'' 
    unpublished report in Comment No. C176, Docket No. 77N-0094, Dockets 
    Management Branch.
        (3) Jones, K., and C. M. Castleden, ``A Double-Blind Comparison 
    of Quinine Sulphate and Placebo in Muscle Cramps,'' Age and Ageing, 
    12(2):155-158, 1983.
        5. Several comments contended that the true incidence of quinine-
    induced thrombocytopenia is many times smaller than that suggested by 
    estimates based on events reported from exposure to quinine-containing 
    drug products alone. The comments contended that such estimates fail to 
    account for the much larger exposure to quinine through beverages. Lavy 
    estimated exposure to quinine in beverages to be about 10 times greater 
    than exposure to quinine in drug products (Ref. 1). An agency search of 
    the medical literature identified only 10 cases of hypersensitivity 
    reactions attributed to quinine in beverages (Refs. 2 through 9). One 
    of these reactions occurred following ingestion of a drug product 
    containing quinine by a person presumed to have been previously 
    sensitized by exposure to beverages (Ref. 5). None of these events was 
    fatal.
        The agency finds that the available information supports the safe 
    use of quinine in beverages such as tonic water and bitter lemon. Given 
    the level of consumption of quinine beverages, there is a scarcity of 
    reported hypersensitivity reactions, even assuming that reactions to 
    food products are vastly underreported.
        Despite these safety data from beverage use, the agency does not 
    consider pooling total consumption and adverse reaction data on quinine 
    from food and drug products to be a legitimate basis to judge the 
    safety of drug products containing quinine. First, there are 
    differences in the quinine exposure levels because quinine is present 
    in much greater amounts in drug products. Second, there is a great 
    disparity in the incidence of reports of hypersensitivity reactions to 
    beverage and drug products.
        The agency considers the appropriate basis on which to estimate the 
    incidence of hypersensitivity to quinine-containing drug products in 
    leg cramp sufferers is to evaluate reports of reactions in the people 
    who take such products at the dose, frequency, and for the duration 
    recommended in product labeling. Adjusting the reported incidence of 
    reactions to drug products by pooling data on beverages erroneously 
    exaggerates the risk of reaction to quinine in beverages, vastly 
    underestimates the risk of reaction to quinine-containing drug 
    products, and contradicts the raw data on these products. Both the 
    number and the severity of reported hypersensitivity reactions to drug 
    products containing quinine raise safety concerns about these products 
    (see section I.B., comment 4).
        The agency considers the virtual absence of reports of reactions to 
    beverages containing small amounts of quinine (i.e., not more than 83 
    parts per million) as support for the safety of such use. Thus, the use 
    of quinine salts in food in accord with conditions described in 21 CFR 
    172.575 is not affected by this final rule on drug products for the 
    treatment and/or prevention of nocturnal leg muscle cramps.
    
    References
    
        (1) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treatment and/or Prevention of Nocturnal leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (2) Belkin, G. A., ``Cocktail Purpura. An Unusual Case of 
    Quinine Sensitivity,'' Annals of Internal Medicine, 66(3):583-586, 
    1967.
        (3) Korbitz, B. C., and E. Eisner, ``Cocktail Purpura. Quinine-
    dependent Thrombocytopenia,'' Rocky Mountain Medical Journal, 
    70(10):38-41, 1973.
        (4) Siroty, R. R., ``Purpura on the Rocks - With a Twist,'' 
    Journal of the American Medical Association, 235(23):2521-2522, 
    1976.
        (5) Elliott, H. L., and D. B. Trash, ``Intravenous Coagulation 
    Induced by Quinine,''Scottish Medical Journal, 24(3):244-245, 1979.
        (6) Murray, J. A. et al., ``Bitter Lemon Purpura,'' British 
    Medical Journal, 2(6204): 1551-1552, 1979.
        (7) Calnan, C. D., and G. A. Caron, ``Quinine Sensitivity,'' 
    British Medical Journal, 2:1750-1752, 1961.
        (8) Cundall, R. D., ``Idiosyncrasy to Quinine in Bitter Lemon,'' 
    British Medical Journal, 1:1638, 1964.
        (9) Callaway, J. L., and W. E. Tate, ``Toxic Epidermal 
    Necrolysis Caused by `Gin and Tonic','' Archives of Dermatology, 
    109:909, 1974.
        6. Four comments submitted five reports (Refs. 1 through 5) of 
    controlled clinical studies of quinine alone or in combination with 
    vitamin E. The agency has reviewed these studies for evidence 
    pertaining to the safety of quinine used to treat and/or prevent 
    nocturnal leg muscle cramps.
        The first study (Ref. 1) was a 10-week, crossover study of 69 
    subjects randomized to either 260 milligrams (mg) quinine sulfate or 
    placebo. Subjects were 26 to 77 years of age, with a mean age of 51. 
    Five adverse reactions to quinine (a 7 percent incidence) were 
    reported. One subject was unable to tolerate the quinine because 
    tinnitus (ringing in the ears) in both ears occurred while taking the 
    study drug. Two additional subjects experienced tinnitus while taking 
    quinine, but continued the medication and completed the study (although 
    the protocol called for discontinuation of the study drug if ringing in 
    the ears occurred). One additional subject experienced disorientation 
    and another reported dizziness while taking quinine. No adverse events 
    to placebo were reported.
        The second study (Ref. 2) was a 10-week, double-blind, randomized, 
    crossover study of 62 subjects receiving daily doses of either 325 mg 
    quinine sulfate or placebo. Subjects were 21 to 76 years of age, with a 
    mean age of 47. Three subjects on quinine and three subjects on placebo 
    reported adverse events. The quinine reactions included tinnitus (which 
    quickly resolved when the drug was discontinued), blurred vision, and 
    headache, which occurred on 3 days during drug administration. One of 
    the subjects dropped out of the study because of dizziness and 
    drowsiness. Another subject discontinued quinine for the last two days 
    of the treatment period because of tinnitus. In the placebo group, one 
    subject reported chest pains and heartburn; another subject experienced 
    fever and nausea; and one subject reported constipation and dropped out 
    of the study. An additional eight subjects dropped out of the study for 
    reasons described in the report as not related to the drug products, 
    but no details were provided.
        The third study (Ref. 3) was a 5-week randomized, crossover study 
    involving 205 subjects randomly assigned to quinine sulfate 260 mg/day, 
    vitamin E 1,600 international units (I.U.)/day, a combination of 
    quinine and vitamin E in the above doses, or placebo. Subjects were 18 
    to 80 years of age, with a mean age of 44. Twenty-seven adverse 
    reactions were reported: 9 (4.4 percent) in subjects receiving the 
    combination of quinine and vitamin E, 8 (3.9 percent) in subjects on 
    quinine alone, 6 (2.9 percent) in subjects on vitamin E alone, and 4 (2 
    percent) in subjects receiving placebo. There was an almost twofold 
    difference in overall adverse experiences when subjects were taking 
    either of the test drugs containing quinine. Adverse events in subjects 
    receiving quinine alone included stomach cramps, headache, nausea, 
    diarrhea, swollen hands, and slight muscle twitching. Adverse events in 
    subjects receiving the quinine and vitamin E combination included upset 
    stomach, headache, diarrhea, tiredness, constipation, and pain in the 
    legs. Adverse effects in subjects receiving vitamin E included 
    abdominal cramps, vomiting, loose bowels, headache, and intensified 
    menstrual cramps. Adverse events in the placebo group included nausea, 
    stomach cramps, and tingly fingers. Gastrointestinal disturbances were 
    reported by twice as many subjects when they were taking quinine alone 
    or in combination than when assigned to vitamin E alone or placebo. 
    Most of the adverse experiences in this study, irrespective of 
    treatment group, were described by investigators as not related or 
    probably not related to the study medication. Reported events, however, 
    were consistent with those classically associated with quinine 
    toxicity, which includes gastrointestinal symptoms (nausea, vomiting, 
    abdominal pain, and diarrhea), vasodilation, sweating, headache, 
    tinnitus, vertigo, and visual disturbances (Ref. 6).
        The fourth study (Ref. 4) was a 5-week, crossover study involving 
    24 subjects 51 to 64 years of age (with a mean age of 57). All subjects 
    received placebo in weeks 1, 3, and 5; half of the subjects were 
    assigned to quinine sulfate (260 mg per (/) day) and half to the 
    combination of quinine sulfate and vitamin E during week 2. In week 4, 
    those subjects on the combination in week 2 were assigned to quinine 
    alone and vice versa. Nausea was reported by 3 subjects (12.5 percent) 
    who received quinine sulfate during week 2. No details of the reported 
    reactions were provided. No other adverse events were reported.
        The fifth and largest study was a multicenter, block-randomized, 
    parallel design involving 559 subjects 18 to 84 years of age (Ref. 5). 
    A 1-week, single-blind, placebo phase was followed by a 2-week, double-
    blind, randomized, treatment phase. Subjects who had at least one leg 
    cramp per night for a minimum of 3 nights during the single-blind 
    placebo week and met other selection criteria were randomized to one of 
    four double-blind treatment groups: Quinine sulfate 259.2 mg (subject 
    ages ranged from 19 to 79 years with a mean age of 46), vitamin E, 
    1,600 I.U. (subject ages ranged from 18 to 76 years with a mean age of 
    42), a combination of quinine and vitamin E in the above doses (subject 
    ages ranged from 18 to 83 years with a mean age of 46), and placebo 
    (subject ages ranged from 21 to 84 years with a mean age of 45). 
    Besides meeting the criteria for frequency of nocturnal leg cramps, 
    subjects admitted to the study were generally in good health, were 
    predominantly female, and had a mean age of less than 50 years. The 
    study report stated that no unexpected or idiosyncratic adverse events 
    were seen ``* * * among patients taking an effective course of therapy 
    * * * nor was there a higher than usual incidence of recognized adverse 
    drug reactions associated with ingestion of quinine.''
        One subject randomized to the combination product was reported to 
    have experienced a reaction consisting of fever, headache, nausea, 
    vomiting, and diffuse muscle pain after 5 days. The episode was 
    sufficiently severe to warrant medical intervention in which the test 
    drug (quinine/vitamin E) was stopped and the subject was treated with 
    analgesic/antipyretic therapy and a prescription antiemetic. Symptoms 
    subsided over 3 days. When the study medication was resumed, the 
    subject again experienced nausea, vomiting, abdominal pain, severe 
    headache, diffuse myalgia with severe pain in the legs, and fever. The 
    subject required hospitalization. Therefore, the study drug was 
    stopped. When the subject was discharged 5 days later, the physician 
    advised her to consider herself sensitive to quinine. Given the 
    temporal relationship between the onset of symptoms and administration 
    of the study drug as well as the positive rechallenge, this case 
    appears to be a well-documented hypersensitivity reaction.
        In addition, another subject on quinine experienced itching, 
    nausea, and vomiting and discontinued the drug. Two other subjects 
    experienced moderately severe wheezing on the 8th and 12th days of 
    quinine treatment, but neither subject discontinued the medication.
        The overall incidence of adverse events reported in the fifth study 
    (Ref. 5) was high and approximately equal in all groups (quinine 
    sulfate, 43.3 percent; vitamin E, 37.2 percent; the combination of 
    quinine sulfate and vitamin E, 39.3 percent; and placebo, 41.3 
    percent). Headache accounted for the greatest number of reported events 
    in each group (quinine sulfate, 19.1 percent; vitamin E, 16.8 percent; 
    combination of quinine sulfate and vitamin E, 19.1 percent; and 
    placebo, 21 percent), but did not appear to be treatment related. 
    Differences in the side- effect profile of the treatments emerge when 
    only events with an incidence of 1 percent or more are considered, and 
    both headache and events considered by the investigators as not related 
    to the study drug are excluded. This analysis shows an adverse event 
    rate of 12.8 percent with quinine sulfate (nausea, vomiting, diarrhea, 
    dizziness, tinnitus, pruritus, and urticaria); 3.6 percent with vitamin 
    E (nausea and myalgia); and 12 percent with the combination product 
    (nausea, vomiting, diarrhea, tinnitus, and fever). None of the events 
    reported by subjects on placebo, which the investigators considered 
    potentially related to the study drug, had an incidence of 1 percent or 
    more. Similarly, events reported to be severe or moderately severe 
    (excluding headache and events with an incidence less than 1 percent) 
    were more frequent in subjects taking quinine sulfate (6.4 percent) or 
    the combination product (7.1 percent) than vitamin E alone (3.6 
    percent) or placebo (2.2 percent).
        The potential for symptoms of quinine toxicity from the low doses 
    generally recommended for the OTC treatment and/or prevention of 
    nocturnal leg muscle cramps has been confirmed in several other studies 
    (Refs. 7, 8, and 9). A recent study of the relationship between plasma 
    quinine levels and hearing impairment (Ref. 7) found that quinine, even 
    at low doses, produced auditory changes. In this study, single oral 
    doses of quinine of 5 milligrams/kilogram (mg/kg), 10 mg/kg, and 15 mg/
    kg were administered to six healthy females 24 to 39 years of age. The 
    study did not specify subject weights. If subject weights of 50 to 60 
    kg were assumed, these doses would correspond to single quinine doses 
    of 250 to 300 mg, 500 to 600 mg, and 750 to 900 mg, respectively. Even 
    at the lowest dose (which is equivalent to the dose used in OTC drug 
    products for the treatment and/or prevention of nocturnal leg muscle 
    cramps), a drug effect on hearing impairment was detected in half of 
    the subjects. When plasma concentrations exceeded 15 micromoles/liter 
    (mmol/L), subjective hearing loss or tinnitus was observed in all 
    subjects. No symptoms were detectable at levels below 5 mmol/L. The 
    shift in hearing threshold was equal over the frequency range studied. 
    The effect over time was consistent with the level of the dose given. 
    The investigators concluded that a consistent effect-concentration 
    relationship for hearing impairment caused by quinine can be defined by 
    audiometry.
        Zajtchuk (Ref. 8) compared the effect on vestibular and auditory 
    function of 0, 52.5 mg, and 105 mg quinine administered in the form of 
    commercial tonic water (containing 52.5 mg of quinine per 822 
    milliliter (mL) bottle) in 17 active duty military personnel. The study 
    was initiated following findings of low levels of quinine in post-
    mortem tissues from military pilot fatalities. Tonic water was 
    administered over a 3-hour period daily for 14 days. While control 
    subjects and subjects given the low dose had normal function throughout 
    the test, three of the four subjects given 105 mg/day developed 
    transient vestibular abnormalities (manifested by rapid, involuntary, 
    rhythmic movements of the eyeball associated with certain positions of 
    the head or body) on positional testing.
        Worden, Shephard, and Frape (Ref. 9) conducted two similar studies. 
    In one study, 6 men and 14 women (18 to 39 years of age) were given 100 
    mg quinine hydrochloride daily in the form of tonic water for 14 days. 
    In the second study, 4 men and 6 women (18 to 53 years of age) were 
    divided into 2 groups. One group received 120 mg in a fortified tonic 
    water, while the other drank a carbonated drink without quinine. No 
    audiometric changes were found in any of the subjects in either study. 
    However, 12 subjects (60 percent) in the first study complained of 
    visual disturbances, 14 (70 percent) reported dizziness, and 14 (70 
    percent) experienced headache.
        The potential for adverse effects from quinine may be greater in 
    the elderly. A survey at one hospital (Ref. 10) of 201 inpatients 70 
    years of age or older found that 23 (11 percent) were taking quinine 
    for cramps. Sixty percent were taking 300 mg nightly; 40 percent were 
    taking twice that amount (600 mg nightly). Approximately one-third of 
    these subjects had been taking the drug continuously and chronically 
    for 2 years or more. The authors noted that the mean elimination half-
    life of quinine in elderly patients has been reported to be 19 hours 
    compared with 8.5 hours in younger adults. The authors also stated that 
    ``Chronic therapy is likely to result in accumulation of quinine, 
    putting elderly patients at greater risk of adverse effects. Possible 
    adverse effects include symptoms of cinchonism (tinnitus, headache, 
    nausea, rash, visual disturbance and temporary blindness), allergic 
    reactions, and thrombocytopenia and haemolytic anaemia.''
        Wanwimolruk et al. (Ref. 11) found the elimination half-life of 
    quinine to be 18.4 plus-minuss> 5.7 hours in 8 healthy elderly 
    subjects 65 to 78 years old compared with 10.5 plus-minuss> 1.6 
    hours in 12 subjects 20 to 35 years old. Furthermore, a significantly 
    greater amount of quinine was excreted unchanged in the elderly 
    subjects, suggesting that quinine metabolism is reduced in elderly 
    people. Overall, there was a 26-percent reduction in clearance of 
    quinine in the older group. The authors concluded that accumulation of 
    quinine may occur in elderly people, thus placing them at a greater 
    risk of adverse events.
        These studies indicate that serious safety concerns exist with 
    regard to the OTC availability of quinine sulfate for the treatment 
    and/or prevention of nocturnal leg muscle cramps. Subjects in all 
    studies submitted were generally in good health, with a mean age of 44 
    to 57 years in the various groups. However, the adverse reactions 
    reported in these studies suggest that quinine doses of 260 to 325 mg/
    day in healthy, middle-aged adults can produce symptoms of quinine 
    toxicity, including auditory, visual, and gastrointestinal effects. 
    Some studies (Refs. 7, 8, and 9) suggest that the vestibular, auditory, 
    visual, and vascular effects of quinine can occur in healthy young 
    adults at doses in and below the range commonly employed for the 
    treatment and/or prevention of nocturnal leg muscle cramps. Altered 
    pharmacokinetics with age also result in a longer half-life of quinine 
    in older people. This longer half-life increases the frequency and 
    severity of adverse effects in the elderly (Ref. 11), a group in which 
    leg cramps are a common condition (Refs. 12 and 13). Therefore, the 
    agency concludes that quinine is not safe for OTC use in the treatment 
    and/or prevention of nocturnal leg muscle cramps.
    
    References
    
        (1) Bottner, M., ``Clinical Trial of the Efficacy of Quinine 
    Sulfate in the Treatment of Nocturnal Leg Muscle Cramps, Protocol 
    84-46,'' draft of an unpublished paper in Comment No. C123, Docket 
    No. 77N-0094, Dockets Management Branch.
        (2) Hays, R., and J. J. Goodman,``Clinical Trial of the Efficacy 
    of Quinine Sulfate in the Treatment of Nocturnal Leg Muscle Cramps, 
    Protocol 86-48,'' draft of an unpublished paper in Comment No. C126, 
    Docket No. 77N-0094, Dockets Management Branch.
        (3) Leo Winter Associates, Inc., ``FinalMedical Report and Data 
    Summary Analysis and Final Statistical Report on Double Blind 
    Randomized Crossover Study of Q-VELR Versus Quinine Sulfate Versus 
    Vitamin E Versus Placebo in the Treatment of Nocturnal Leg Muscle 
    Cramps (No. 1285-5082),'' draft of an unpublished paper in Comment 
    No. SUP00031, Docket No. 77N-0094, Dockets Management Branch.
        (4)Biodesign GmbH, ``Clinical Evaluationof Q-VELR in Patients 
    with Nocturnal Leg Muscle Cramps,'' draft of an unpublished paper in 
    Comment No. SUP00031, Docket No. 77N-0094, Dockets Management 
    Branch.
        (5) Draft of an unpublished study entitled: ``A Short-Term, 
    Randomized, Double-Blind, Parallel Study of Q-Vel vs. Quinine 
    Sulfate vs. Vitamin E vs. Placebo in the Prevention and Treatment of 
    Nocturnal Leg Cramps,'' Comment No. SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (6) Bateman, D. N., and E. H. Dyson, ``Quinine Toxicity,'' 
    Adverse Drug Reactions and Acute Poisoning Reviews, 4:215-233, 1986
        (7) Alvan, G. et al., ``Hearing Impairment related to Plasma 
    Quinine Concentration in Healthy Volunteers,``British Journal of 
    Clinical Pharmacology, 31:409-412, 1991.
         (8) Zajtchuk, J. T. et al., ''Electronystagmographic findings 
    in Long-term Low-Dose Quinine Ingestion,`` Archives of 
    Otolaryngology, 110:788-791, 1984.
        (9) Worden, A. N., N. W. Shephard, and D. L. Frape, ``Report on 
    the Daily Consumption for 14 Days by Normal Subjects of Tonic Water 
    Containing Quinine Hydrochloride,'' unpublished report, copy in OTC 
    Vol. 03AFM, Docket No. 77N-0094, Dockets Management Branch.
        (10) Blackbourn, J., and D. Bajrovic, ``Quinine--Forever and 
    Ever?,'' Hospital Pharmacy, 23:732, 735, 1988.
        (11) Wanwimolruk, S. et al., ``Pharmaco-kinetics of Quinine in 
    Young and Elderly Subjects,'' Transactions of the Royal Society of 
    Tropical Medicine and Hygiene, 85(6):714-717, 1991.
        (12) Griggs, R. C., ``Pain, Spasm, and Cramps of Muscle,'' in 
    ``Harrison's Principles of Internal Medicine,'' 12th ed., edited by 
    J. D. Wilson et al., McGraw-Hill, Inc., New York, pp. 173-176, 1991.
        (13) Jones, K., and C. M. Castleden, ``A Double-Blind Comparison 
    of Quinine Sulphate and Placebo in Muscle Cramps,'' Age and Ageing, 
    12(2):155-158, 1983.
        7. One comment (Ref. 1) requested a ban on the OTC marketing of any 
    quinine sulfate products used in the treatment of nocturnal recumbency 
    leg cramps. The comment based this request, in part, on adverse 
    reactions reported to the FDA, including eight deaths it described as 
    closely linked to quinine products. The comment contended that serious 
    and fatal adverse reactions to quinine sulfate purchased OTC for this 
    use continue to be reported. The comment mentioned that these reactions 
    can occur in several ways: (1) In persons hypersensitive to quinine, 
    (2) from the innate toxicity of quinine, or (3) as a result of 
    interactions with other drugs, including digoxin, anticoagulants, and 
    antiarrhythmics. The comment concluded that the risks associated with 
    quinine used for leg cramps are unacceptable in light of its lack of 
    efficacy for this use.
        The agency agrees that quinine has the potential to elicit serious 
    hypersensitivity reactions at doses employed in OTC drug products used 
    for the treatment and/or prevention of nocturnal leg muscle cramps. The 
    agency's spontaneous adverse reaction reporting system includes 
    reasonably unconfounded reports of thrombocytopenia, hemolytic anemia, 
    leukopenia, granulocytopenia, anaphylaxis, hypersensitivity syndrome, 
    severe skin reactions (including urticaria, angioedema, and erythema 
    multiforme), liver abnormalities, renal failure, and death (see section 
    I.B., comment 4). Reports in the literature have identified quinine 
    sulfate (in doses typically recommended for the treatment and/or 
    prevention of nocturnal leg muscle cramps) as the causative agent in 
    cases of photosensitive dermatitis (Refs. 2, 3, and 4), psychosis (Ref. 
    5), disseminated intravascular coagulation (Ref. 6), and hemolytic 
    uremic syndrome (Ref. 7).
        The agency agrees that quinine may interact with several other 
    drugs (Refs. 8, 9, and 10), including those mentioned by the comment. 
    This information could be included in the labeling of OTC quinine drug 
    products. However, the agency does not need to make a decision on such 
    drug interaction precautions because no ingredients for treating and/or 
    preventing nocturnal leg muscle cramps are currently generally 
    recognized as safe and effective for inclusion in an OTC drug 
    monograph.
        Cinchonism is a cluster of symptoms of varying severity that 
    includes: Tinnitus, dizziness, disorientation, nausea, visual changes, 
    auditory deficits, and (at higher doses) cardiac arrhythmias. 
    Cinchonism is dose related. The clinical studies discussed in section 
    I.B., comment 6 demonstrate that adverse events typical of quinine 
    toxicity (in some cases, sufficiently severe to lead to discontinuation 
    of the drug) occur in some people at doses generally recommended for 
    the treatment and/or prevention of nocturnal leg muscle cramps. These 
    studies indicate that some people who self-medicate with quinine to 
    treat and/or prevent nocturnal leg muscle cramps at doses recommended 
    in product labeling will experience these quinine-related adverse 
    events. In addition, people taking quinine remain at risk of developing 
    hypersensitivity to the drug and experiencing a serious, life 
    threatening, or fatal reaction as a consequence. Even if quinine were 
    effective for the treatment and/or prevention of nocturnal leg muscle 
    cramps, this risk would require that a prescribing physician 
    participate in the decision to use the drug, by assuring the diagnosis, 
    considering alternative treatment options, evaluating concurrent 
    medical problems and medications, and monitoring patient safety 
    throughout treatment.
    
    References
    
        (1) Comment No. CP0006, Docket No. 77N-0094, Dockets Management 
    Branch.
        (2) Diffey, B. L. et al., ``The Action Spectrum in Quinine 
    Photosensitivity,'' British Journal of Dermatology, 118:679-685, 
    1988.
        (3) Ferguson, J. et al., ``Quinine Induced Photosensitivity: 
    Clinical and Experimental Studies,'' British Journal of Dermatology, 
    117:631-40, 1987.
        (4) Ljunggren, B., and P. Sjovall, ``Systemic Quinine 
    Photosensitivity,'' Archives of Dermatology , 122:909-911, 1986.
        (5) Verghese, C., ``Quinine Psychosis,'' British Journal of 
    Psychiatry, 153:575-576, 1988.
        (6) Spearing, R. L. et al., ``Quinine-induced Disseminated 
    Intravascular Coagulation,'' Lancet, 336:1535-1537, 1990.
        (7) Gottschall, J. L. et al., ``Quinine-Induced Immune 
    Thrombocytopenia Associated with Hemolytic Uremic Syndrome: A New 
    Clinical Entity,'' Blood, 77(2):306-310, 1991.
        (8) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treatment and/or Prevention of Nocturnal Leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (9) USPDI, ``Drug Information for the Health Care 
    Professional,'' The U. S. Pharmacopeial Convention, Inc., Rockville, 
    MD, vol. I, 14th ed., pp. 2379-2382, 1994.
        (10) ``Drug Evaluations Subscription,'' American Medical 
    Association, Chicago, 1 (4):10-11, 1993.
        8. Several comments downplayed the potential for hypersensitivity 
    reactions to quinine, particularly quinine- induced immunologic 
    thrombocytopenia, arguing that the continued OTC marketing of quinine 
    for the treatment and/or prevention of nocturnal leg muscle cramps 
    should not be stopped because of this potential consequence of its use. 
    One comment (Ref. 1) submitted an expert review of drug-induced 
    immunologic thrombocytopenia (DIITP), which stated that DIITP has been 
    reported with over 100 drugs. Gold salts, heparin, and the cinchona 
    alkaloids are the drugs most commonly associated with this condition. 
    According to the expert review supplied by the comment, there is no 
    known information about the dose of quinine required to induce DIITP 
    sensitivity. DIITP occurs more frequently in people over 50 years old, 
    possibly because of their greater exposure to drugs. It typically is 
    characterized by a warm sensation, followed by a chill. Bleeding 
    episodes, manifested by petechiae (pinpoint red spots, caused by 
    intradermal or submucosal hemorrhage), purpura (purplish or brownish 
    red discolorations visible through the skin, caused by hemorrhage into 
    the tissues), hemorrhagic lesions of the oral mucosa, and occasionally 
    hemorrhage of the gastrointestinal and urinary tracts may occur 6 to 12 
    hours after drug exposure in individuals who develop severe 
    thrombocytopenia. Intracerebral hemorrhage and lethal intrapulmonary 
    hemorrhage have been reported. Primary treatment is to discontinue the 
    offending drug; bleeding usually subsides in 3 to 4 days. Other 
    interventions (including glucocorticoid therapy and platelet 
    transfusions) have not been shown to be beneficial.
        Another comment argued that many drugs and additives to foods have 
    the propensity to induce a variety of adverse reactions (Ref. 2). The 
    comment stated that the prevalence of hypersensitivity to tartrazine 
    (FD&C Yellow No. 5), a widely used dye, has been estimated to be about 
    1 in 10,000 in the general population. The comment pointed out that 
    when tartrazine is used in OTC drug products, a labeling statement is 
    required to inform consumers that the product contains tartrazine and 
    that it may cause allergic-type reactions. The comment stated that this 
    was ``a clear precedent for the OTC drug use of products that have 
    potential for rare hypersensitivity.'' The comment also described 
    aspirin sensitivity as widespread and emphasized that a brief warning 
    statement in labeling regarding use by people with asthma or aspirin 
    sensitivity is deemed adequate to ensure safe OTC use.
        The agency finds that the information in the first comment 
    indicates that quinine is one of the drugs most frequently associated 
    with DIITP. While other drugs (e.g., gold salts and heparin) cause 
    DIITP, quinine is the only drug highly associated with DIITP that is 
    available OTC.
        In March 1985, the Department of Health and Human Services 
    established an Ad Hoc Advisory Committee on Hypersensitivity to Food 
    Constituents (the Committee) to evaluate data relevant to allergic-type 
    reactions in humans that were associated with food constituents. The 
    Committee concluded that tartrazine may cause mild cases of urticaria 
    (hives) in a small subset of the population (usually not requiring 
    medical intervention). The Committee found no evidence that the color 
    additive constitutes a hazard to the general public when used in food 
    at its current levels. Prior to the Committee's findings, the agency 
    had decided that labeling provides an adequate safeguard for those 
    sensitive to tartrazine. (See the Federal Register of February 4, 1977 
    (42 FR 6835) and June 26, 1979 (44 FR 37212).) The agency requires the 
    label of OTC and prescription drug products containing tartrazine 
    intended for oral, nasal, rectal, or vaginal use to specifically 
    declare the presence of tartrazine by listing the color additive using 
    the names ``FD&C Yellow No. 5'' and ``tartrazine.'' (See 21 CFR 
    74.1705(c)(2).) In addition to this label statement, prescription drug 
    products for these uses must also include in their labeling the warning 
    statement ``This product contains FD&C Yellow No. 5 (tartrazine) which 
    may cause allergic-type reactions (including bronchial asthma) in 
    certain susceptible persons. Although the overall incidence of FD&C 
    Yellow No. 5 (tartrazine) sensitivity in the general population is low, 
    it is frequently seen in patients who also have aspirin 
    hypersensitivity.''
        There are a number of differences between hypersensitivity 
    reactions to tartrazine and aspirin and hypersensitivity reactions to 
    quinine. In a review of allergic reactions to drug additives (Ref. 3), 
    Simon stated that reactions to tartrazine ``if they occur at all, are 
    indeed quite rare for the asthmatic population, even for the aspirin-
    sensitive subpopulation.'' Simon further reported that no positive 
    responses were found after 125 double-blind, placebo-controlled 
    tartrazine challenges (with at least 25 mg) in an aspirin-sensitive 
    asthmatic population. Simon also reviewed adverse reactions to food 
    additives (Ref. 4) and stated that, although tartrazine is the food 
    additive most frequently associated with hypersensitivity reactions, 
    ``tartrazine has been confirmed to be at best only occasionally 
    associated with flares of urticaria or asthma.'' Reports of these 
    relatively mild tartrazine reactions, however, are in contrast to the 
    serious reports for quinine, which involve life threatening and fatal 
    hypersensitivity reactions.
        Virchow et al. (Ref. 5) evaluated sensitivity to tartrazine in 156 
    Europeans with confirmed aspirin-induced asthma. Oral challenges were 
    performed with increasing doses. All positive challenges were repeated 
    under double-blind conditions. Only four subjects had positive 
    reactions; none were serious. The incidence of tartrazine cross 
    sensitivity to aspirin in this European population was 2.6 percent. In 
    a similar study, Morales et al. (Ref. 6) conducted 141 challenge tests 
    on 47 subjects with asthma associated with intolerance to analgesics, 
    using tartrazine doses of 5, 25, 50, 100, and 200 mg and a placebo. 
    Only five tests were positive in four of the subjects; repeat tests 
    were negative in three of the four subjects. The authors stated that 
    clinical instability in these subjects may be the cause of some 
    respiratory symptoms attributed to tartrazine and that the practice of 
    recommending color free diets should be reserved for cases in which a 
    positive challenge test has been obtained on at least two occasions. 
    This experience suggests that: (1) The incidence of tartrazine 
    sensitivity may be overestimated, and (2) the nature of reactions to 
    tartrazine is sufficiently benign to permit multiple rechallenges to 
    confirm intolerance. Rechallenge of quinine-sensitive individuals, in 
    contrast, is contraindicated because the reactions are serious, life 
    threatening, or fatal, even under controlled conditions.
        Safford (Ref. 7) was unable to detect antibody formation with 
    tartrazine and its metabolites in animal studies, suggesting that an 
    immunologic response is not involved in tartrazine sensitivity. 
    Hypersensitivity to quinine, in contrast, is mediated by an immunologic 
    mechanism.
        Aspirin sensitivity is relatively common compared to quinine 
    sensitivity, but is more manageable and usually predictable. In a 
    review of aspirin sensitivity, Settipane (Ref. 8) described a number of 
    factors that are predictive of subjects in whom intolerance is most 
    likely to occur. Sensitivity is seen in 23 to 28 percent of people with 
    chronic urticaria, 14 to 23 percent of people with nasal polyps, and up 
    to 19 percent of people with asthma. These people are likely to be 
    under a doctor's care and to have been told to avoid aspirin products. 
    Genton et al. (Ref. 9) studied the usefulness of oral provocation tests 
    to aspirin and food additives in 34 subjects with asthma or chronic 
    urticaria, concluding that such investigations are safe and useful in 
    managing such subjects by identifying intolerance to various compounds. 
    As with tartrazine, hypersensitivity to aspirin does not appear to be 
    mediated by an immunologic response (Ref. 8). In contrast to aspirin, 
    there are no predictive factors for quinine hypersensitivity and, as 
    noted above, in vivo rechallenge is contraindicated.
        Sensitivity to aspirin (Ref. 8) and tartrazine (Ref. 10) is a 
    problem that is manageable. The sensitivity generally results in 
    urticarial or bronchospastic symptoms that are responsive to medical 
    treatment. Anaphylaxis has been reported with aspirin, but is extremely 
    rare given the extensive use of products containing aspirin. In a 
    retrospective study of anaphylaxis occurring outside of hospitals in a 
    hospital catchment area in Denmark over a 13-year period, the rate of 
    anaphylaxis caused by aspirin was 0.48 cases per 100,000 inhabitants 
    (Ref. 11). Sensitivity to quinine, in contrast to aspirin or 
    tartrazine, affects a number of body systems and may be serious, 
    manifested as urticaria/angioedema, hepatic injury, renal failure, 
    serious dermatologic conditions, serious hematologic events, and death 
    (Ref. 12) (also see section I.B., comment 6). Three sources estimate 
    the incidence of quinine-induced immunologic thrombocytopenia to be in 
    the range of about 1:1,000 to 1:3,000 (see section I.B., comment 9).
        FDA's spontaneous reporting system contains 110 case reports 
    involving quinine for the period from 1969 through 1990. Sixty-nine 
    (approximately 63 percent) of these reports represent possible 
    hypersensitivity reactions, including 22 reports of thrombocytopenia 
    (57 of these cases [approximately 83 percent] involved quinine products 
    and/or quinine dosages typically used in the treatment and/or 
    prevention of nocturnal leg muscle cramps). Of the eight deaths that 
    occurred among the reported hypersensitivity reactions, medical records 
    and autopsy findings were sufficiently complete in two of these cases 
    (both involving OTC quinine products indicated for the treatment of leg 
    muscle cramps) to implicate quinine-induced thrombocytopenia as 
    precipitating fatal hemorrhages in each case. Underreporting of such 
    reactions into the agency's spontaneous reporting system is believed to 
    be very substantial for OTC drug products. This may be due to 
    physicians (the principal reporters to the spontaneous reporting 
    system) not becoming aware of reactions to OTC drugs, and because 
    manufacturers and distributors are not generally required to transmit 
    reports of serious adverse reactions involving OTC drugs to FDA.
        The agency concludes that the severity of quinine hypersensitivity 
    reactions, even in their first occurrence, and the inability to 
    identify predisposing factors to this occurrence create a risk clearly 
    different from that presented by tartrazine or aspirin. The agency does 
    not consider it likely that a warning statement in quinine product 
    labeling would be of significant value because it is impossible to 
    prospectively identify the groups at risk (see section I.B., comment 
    10).
    
    References
    
        (1) Aster, R. H., ``Thrombocytopenia Induced by Quinine and 
    Other Drugs'', unpublished report in Comment No. C143, Docket No. 
    77N-0094, Dockets Management Branch.
        (2) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treatment and/or Prevention of Nocturnal Leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (3) Simon, R. A., ``Adverse Reactions to Drug Additives,'' 
    Journal of Allergy and Clinical Immunology, 74(4 Pt 2):623-630, 
    1984.
        (4) Simon, R. A., ``Adverse Reactions to Food Additives,'' New 
    England and Regional Allergy Proceedings, 7(6):533-542, 1986.
        (5) Virchow, C. et al., ``Intolerance to Tartrazine in Aspirin-
    Induced Asthma: Results of a Multicenter Study,'' Respiration, 
    53(1):20-23, 1988.
        (6) Morales, M. C. et al., ``Challenge Tests with Tartrazine in 
    Patients with Asthma Associated with Intolerance to Analgesics (ASA-
    Triad): A Comparative Study with Placebo,`` Clinical Allergy, 
    15(1):55-59, 1985.
        (7) Safford, R. J., and B. F. Goodwin, ''Immunological Studies 
    on Tartrazine and its Metabolites. I. Animal Studies.'' 
    International Archives of Allergy and Applied Immunology, 77(3):331-
    336, 1985.
        (8) Settipane, G. A., ``Aspirin and Allergic Diseases: A 
    Review,''American Journal of Medicine, 74(6A):102-109, 1983.
        (9) Genton, C., P. C. Frei, and A. Pecoud, ``Value of Oral 
    Provocation Tests to Aspirin and Food Additives in the Routine 
    Investigation of Asthma and Chronic Urticaria,`` Journal of Allergy 
    and Clinical Immunology, 76(1):40-45, 1985.
        (10) Dipalma, J. R., ''Tartrazine Sensitivity,'' American Family 
    Physician, 42(5):1347-1350, 1990.
        (11) Sorensen, H. T., B. Nielsen, and J.Ostergaard-Nielsen, 
    ``Anaphylactic Shock Occurring Outside Hospitals,'' Allergy, 
    44(4):288-290, 1989.
        (12) Bateman, D. N., and E. H. Dyson,``Quinine Toxicity,'' 
    Adverse Drug Reactions and Acute Poisoning Reviews, 4:215-233, 1986.
        9. Two comments provided estimates of the incidence of quinine-
    induced immunologic thrombocytopenia (QIITP). Lavy (Ref. 1) presented 
    several estimates, each based on different assumptions and information. 
    For one estimate, Lavy noted that four of six hypersensitivity 
    reactions reported to FDA in 1987 were cases of thrombocytopenia. Lavy 
    converted the total sales of quinine drug products for 1987 by dosage 
    unit to ``total number of days of therapy sold'' by dividing the number 
    of tablets and capsules sold by the dose per day described in product 
    labeling. Lavy assumed that quinine was taken for leg cramps 
    approximately one quarter of the year by each subject. By dividing the 
    ``total days of therapy purchased'' by the ``total days used per 
    person,'' Lavy estimated the size of the population exposed to drug 
    products containing quinine in 1987 to be 1.66 x 106, and calculated 
    the incidence of QIITP to be 1 case per 415,000 people, based upon 4 
    cases reported to FDA that year. Lavy did not try to correct for 
    underreporting.
        Using another approach, Lavy reported that quinine has been 
    estimated to be the causative agent in approximately 10 percent of all 
    drug-induced immunologic thrombocytopenia reports. He noted that 
    secondary thrombocytopenia was the principal diagnosis in approximately 
    4,000 discharges in the 1987 National Hospital Discharge Survey. 
    Assuming that 10 percent of these thrombocytopenia cases were drug-
    induced and 10 percent of drug-induced immunologic thrombocytopenia 
    cases are related to quinine, 40 cases could be attributed to quinine. 
    Assuming the population exposed to drug products containing quinine in 
    1987 was 1.66 x 106 (as calculated above), Lavy calculated the 
    incidence of QIITP to be 1:41,500. Lavy cited a third estimate of the 
    incidence of QIITP based on information from Danielsen's report on 
    drug-induced blood disorders among admissions at the Group Health 
    Cooperative of Puget Sound (Ref. 2). In this retrospective study, 6 
    cases of thrombocytopenia related to quinine or quinidine among 5,089 
    subjects were reported for an apparent incidence of 1 case per 848 
    subjects taking 1 or the other of the 2 drugs.
        Another comment (Ref. 3) estimated the incidence of QIITP from 
    ingestion of drug products to be 1:3,300 per year. The comment based 
    its calculations on the number of cases of documented quinine-induced 
    thrombocytopenia at the Blood Center of Southeastern Wisconsin over a 
    10-year period. In making this estimate, it was assumed that at least 
    half of all cases occurring in this population would have been referred 
    to the laboratory for confirmation of diagnosis.
        The agency notes that the estimates of the incidence of 
    thrombocytopenic reactions to drug products containing quinine range 
    from more than 1 in 1,000 (for quinine and quinidine considered 
    together) to less than 1 in 400,000. This wide range suggests that a 
    precise estimate will be hard to obtain. It is difficult to conclude, 
    however, that the first estimate proposed by Lavy is correct. The 
    number of events used by Lavy is the number reported to FDA in 1987. 
    While no one knows the extent of underreporting, it is believed to be 
    very substantial. For example, if even a 1 percent rate is assumed, 
    this would translate, using Lavy's other figures, to about 1 in 4,000 
    people. The exposure estimate could also be considerably in error. Lavy 
    assumed the drug was used for one-quarter of the year by each person. 
    If, in fact, it was used for one half of the year, the number of 
    exposed people would be half that proposed and the rate of drug-induced 
    immunologic thrombocytopenia would be double that calculated.
        The incidence calculated based on the National Hospital Discharge 
    Survey (Ref. 1) employed the estimate of population discussed above and 
    assumed 1 percent of the diagnoses of secondary thrombocytopenia were 
    attributable to quinine. There is no way to know the accuracy of this 
    estimate; if it were higher, even by a factor of 5, the estimated rate 
    would be above 1 in 10,000, a substantial rate.
        Probably the most credible of Lavy's estimates is the Puget Sound-
    based estimate (Ref. 2), because it is based on hospital diagnoses and 
    well-documented exposure. The estimate of the incidence of QIITP based 
    on the number of documented cases occurring in the population served by 
    the Blood Center of Southeastern Wisconsin over a 10-year period (Ref. 
    3) also is based on relatively few assumptions and appears reliable. 
    The only assumption in this calculation was that twice as many events 
    occurred as were reported to the laboratory. The estimates from these 
    two sources, 1:848 (Puget Sound) and 1:3,300 (Southeastern Wisconsin) 
    are similar to the estimate of 1:1,000 cited by Mitchell (Ref. 4). 
    These three sources provide a reasonably small range for the incidence 
    of QIITP that can be expected, about 1:1,000 to 1:3,000.
        Therefore, while the agency believes that a precise estimate of the 
    incidence of QIITP will be difficult to obtain, credible estimates from 
    three sources (Refs. 2, 3, and 4) do not support the assertion that 
    QIITP is a rare event.
    
    References
    
        (1) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treat ment and/or Prevention of Nocturnal Leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (2) Danielson, D. A. et al., ``Drug-induced blood disorders,'' 
    Journal of the American Medical Association, 252(23):3257-3260, 
    1984.
        (3) Aster, R. H., ``Thrombocytopenia Induced by Quinine and 
    Other Drugs,'' unpub-lished report in Comment No. C143, Docket No. 
    77N-0094, Dockets Management Branch.
        (4) Mitchell, T. R., and J. D. Morrow, ``Quinine Purpura,'' 
    Journal of the Tennessee Medical Association, 81(9):578, 1988.
        10. Three comments contended that warnings in product labeling 
    could adequately inform and protect consumers from the well-known side 
    effects of quinine, including idiosyncratic reactions. One comment 
    stated that warnings recommended by the Miscellaneous Internal Panel 
    (47 FR 43562 at 43564), including those concerning idiosyncratic 
    reactions, have been incorporated into the labeling of currently 
    marketed products. Another comment stated that careful warning language 
    in its product labeling helps to further protect consumers by informing 
    them of the possibility of untoward, idiosyncratic reactions. This 
    labeling states: ``Discontinue use and consult your doctor immediately 
    if swelling, bruising, skin rash, skin discoloration or bleeding 
    occurs. These symptoms may indicate a serious condition. Discontinue 
    use if ringing in the ears, deafness, diarrhea, nausea or visual 
    disturbances occur * * * Do not take if * * * allergic or sensitive to 
    quinine or under 12 years of age.'' A third comment, citing a report by 
    Lavy (Ref. 1), stated that serious adverse effects occur at a frequency 
    probably less than 1 in 40,000 people (see section I.A., comment 9), 
    the clinical course is only rarely complicated, and that labeling can 
    clearly and concisely warn ``* * * regarding the more common yet low 
    frequency side effects, which are generally treated simply by 
    discontinuing use.''
        In the tentative final monograph (50 FR 46588 at 46592) the agency 
    proposed the following warning in Sec. 343.150(c) for OTC drug products 
    containing quinine: ``Discontinue use if ringing in the ears, deafness, 
    skin rash, or visual disturbances occur. Do not take if pregnant, 
    sensitive to quinine, or under 12 years of age.'' The agency proposed 
    this labeling in the event that data were submitted that resulted in 
    the inclusion of quinine in a monograph in the final rule. While 
    proposed, this labeling was not required at that time.
        The agency has reviewed the warning information currently appearing 
    on OTC quinine products marketed for the treatment and/or prevention of 
    leg muscle cramps. The language varies slightly between products, but 
    the information provided is similar. In general, labeling warns 
    patients to discontinue taking the drug should any of a number of 
    listed events occur. However, the labeling differs in the events listed 
    and in recommending when a physician should be contacted.
        There are several factors that argue against the sufficiency of 
    label warnings to protect consumers from serious adverse events related 
    to quinine. The frequency of these reactions is probably greater than 
    assumed by the comments (see section I.B., comment 9). Many of the 
    adverse advents are unpredictable. For example, thrombocytopenia may 
    occur after 1 week of exposure or after months or years of quinine 
    administration. Further, there may be no characteristic that would 
    predict an adverse event in the person using the product. The agency 
    believes that a physician could help people using this drug appreciate 
    the nature and frequency of the risk and help in the consideration 
    whether that risk is acceptable. The physician could also advise about 
    the signs of thrombocytopenia, such as petechiae (pinpoint, nonraised, 
    round, purplish red spots) and purpura (small hemorrhage), perhaps 
    allowing identification of this condition before a significant 
    hemorrhage occurred. A number of the adverse reaction reports note the 
    occurrence of a similar prior event related to previous ingestion of 
    quinine in which neither the user nor the physician recognized the 
    relationship of the illness to quinine ingestion. Use of quinine under 
    a physician's prescription, with appropriate emphasis on warning signs, 
    may make timely recognition easier.
        Although drug-induced immunologic thrombocytopenia may be the best 
    studied idiosyncratic reaction caused by quinine (Ref. 2), quinine has 
    also been reported to have been associated with a number of other 
    hypersensitivity reactions and pharmacologic effects. Lavy (Ref. 1) 
    notes that these include ``the possibility of decreased digoxin 
    clearance, increased half-life of quinine when given concurrently with 
    cimetidine, pseudo-allergic reactions in aspirin-sensitive patients, 
    drug fever, nonspecific granulomatous hepatitis, asthma, hemolytic 
    anemia, inhibition of tolbutamide metabolism, hypoprothrombinemia, 
    hemolytic anemia in glucose-6-phosphate dehydrogenase (G-6-PD) 
    deficient patients, etc.'' Cooper and Bunn (Ref. 3) reported that G-6-
    PD-deficient individuals (i.e., those variants susceptible to hemolytic 
    anemia from quinine) are relatively common among eastern Mediterranean 
    and Chinese people. Quinine may also interact with several other drugs 
    (see section I.B., comment 7). Furthermore, the possible pharmacologic 
    effects may have particular significance for the elderly who may be 
    taking concomitant medications that either provoke muscle cramps or 
    adversely interact with quinine. Altered pharmacokinetics with age also 
    result in a longer half-life of quinine in older people, which suggests 
    that the frequency and severity of adverse effects may be greater in 
    the elderly (Ref. 4) (also see section I.B., comment 6). The foregoing 
    possible additional adverse reactions, including those related to 
    ethnicity, age, and concurrent drug therapy, are not addressed by the 
    labeling of the comment's product and would generally be difficult to 
    address in OTC drug product labeling.
        It should also be noted that the number of reports of serious 
    adverse reactions submitted to FDA's spontaneous reporting system, 
    including those resulting in hospitalization and death, has been 
    increasing over the past several years in spite of the industry's 
    revision of labeling to incorporate the warnings suggested by the 
    Miscellaneous Internal Panel in 1982. There has been an increasing 
    number of reports per year since 1986, and 56 of 105 reports 
    (approximately 53 percent) have been received by FDA since 1988. (See 
    section I.B., comment 4.)
        The agency concludes there is insufficient evidence that warnings 
    in product labeling could adequately inform and protect consumers from 
    the well-known side effects of quinine, including idiosyncratic 
    reactions. This conclusion is based primarily on the severity of 
    hypersensitivity reactions to drug products that contain quinine and 
    the inability to identify predisposing factors to these reactions, the 
    frequency of such reactions, and the relationship of quinine-related 
    adverse events to factors such as ethnicity, age, and concurrent drug 
    therapy.
    
    References
    
        (1) Lavy, N. W., ``Overview: Efficacy and Safety of Quinine 
    Sulfate in the Treat-ment and/or Prevention of Nocturnal Leg 
    Cramps'', unpublished report in SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (2) Aster, R. H., ``Q-Vel: Could Serious Adverse Reactions be 
    Prevented by Having the Drug Available Only on Prescription,'' 
    unpublished report in Comment No. C176, Docket No. 77N-0094, Dockets 
    Management Branch.
        (3) Cooper, R. A., and H. F. Bunn, ``Hemolytic Anemias,'' in 
    ``Harrison's Principles of Internal Medicine,'' 12th ed., edited by 
    J. D. Wilson et al., McGraw-Hill, New York, pp. 1531-1543, 1991.
        (4) Blackbourn, J., and D. Bajrovic, ``Quinine--Forever and 
    Ever?,'' Hospital Pharmacy, 23:732, 735, 1988.
        11. Two comments objected to the agency's discussion on the safety 
    of vitamin E (50 FR 46588 at 46591), contending that a considerable 
    body of data demonstrating safety in humans had been excluded from the 
    agency's evaluation. The comments primarily objected to the agency's 
    emphasis on the observations of one physician as an expert on vitamin E 
    because they considered the data referred to by this individual to be 
    anecdotal, uncontrolled, and largely subjective. The comments provided 
    a literature review and other data (Refs. 1, 2, and 3) to support the 
    safe use of vitamin E in humans.
        Another comment disagreed with the agency's Category III 
    classification of vitamin E both individually and in combination with 
    quinine sulfate for the treatment and prevention of nocturnal leg 
    muscle cramps, contending that adequate information already existed to 
    support the safety of these ingredients (alone or in combination). The 
    comment included the results of two new clinical studies (Refs. 4 and 
    5) comparing vitamin E, quinine sulfate, a combination product 
    containing vitamin E and quinine sulfate, and placebo to support the 
    safe use of the individual ingredients as well as the combination of 
    these ingredients for this indication. In both studies, subjects 
    received a daily dose of 1,600 I.U. of vitamin E, either alone or in 
    combination with quinine sulfate.
        One additional comment included the results of a third new clinical 
    study comparing vitamin E, quinine sulfate, a combination product 
    containing vitamin E and quinine sulfate, and placebo (Ref. 6). Some 
    safety information on vitamin E can be derived from this study.
        In the tentative final monograph, the agency classified vitamin E 
    in Category III for the treatment and prevention of nocturnal leg 
    muscle cramps, stating that a safe and effective OTC dosage had not 
    been established for this use (50 FR 46588 at 46591). The agency 
    evaluated all of the data that had been submitted to this rulemaking 
    proceeding but acknowledges that these data were not the total body of 
    information that has been published on vitamin E. The agency did point 
    out that the paper by Roberts (Ref. 7) raised some questions about a 
    safe dose of OTC vitamin E.
        The agency has reviewed the additional data and information that 
    have been submitted and determined that sufficient evidence has been 
    presented to support the safety of vitamin E for the treatment and/or 
    prevention of nocturnal leg muscle cramps. However, the evidence is 
    inadequate to support the effectiveness of vitamin E for this use (see 
    section I.C., comment 13).
        Farrell and Bieri (Ref. 2) evaluated potential toxic and/or 
    beneficial effects of vitamin E intake. Twenty-eight adults who had 
    been self-administering 100 to 800 I.U. of vitamin E daily for an 
    average of 3 years were studied. A review of the subjects' past medical 
    histories did not reveal any apparent gross evidence of toxicity from 
    vitamin E intake. The highest plasma alpha-tocopherol concentrations in 
    the vitamin E subjects were two times the upper limit of normal, as 
    determined in control subjects. A broad range of laboratory tests were 
    performed to assess toxic effects on various organ systems. No 
    disturbance in liver, kidney, muscle, thyroid gland, erythrocytes, 
    leukocytes, coagulation parameters, or blood glucose was found.
        Salkeld (Ref. 1) reviewed over 9,000 cases in which daily doses of 
    up to 3,000 I.U. of vitamin E were taken for up to 11 years (and 55,000 
    I.U. daily for 5 months in a few subjects). In 1,014 cases with vitamin 
    E intake from 200 I.U. up to 3,000 I.U. daily for up to 11 years, it 
    was stated that no side effects were observed. In another 8,241 cases 
    with similar intake and duration, there was no mention of side effects. 
    In other trials, 82 of 813 subjects complained of one or more side 
    effects. The reported effects included dermatitis, pruritus ani, acne, 
    cheilosis, fatigue and weakness, gastrointestinal symptoms, prostatic 
    obstruction, tachycardia, and vasodilation. Thus, in a total of 10,068 
    cases, Salkeld found a 0.8 percent overall incidence of side effects. 
    The Advisory Review Panel on OTC Vitamin, Mineral, and Hematinic Drug 
    Products relied in part on this same literature review by Salkeld in 
    stating its conclusion ``that vitamin E is safe'' (March 16, 1979, 44 
    FR 16126 at 16172). The Advisory Review Panel on OTC Antimicrobial (II) 
    Drug Products, in the advance notice of proposed rulemaking for OTC 
    topical acne drug products (March 23, 1982, 47 FR 12430), mentioned 
    that there are no notable pharmacological or toxicological effects of 
    oral vitamin E and that numerous experiments indicate that high dietary 
    intakes of vitamin E (up to 800 I.U. daily for up to 3 years) are 
    apparently without toxic side effects (47 FR 12462).
        One of the new clinical studies submitted includes the results of 
    laboratory tests performed in 24 patients to evaluate the effect of the 
    product on various organ systems (Ref. 4). Tests were performed at 
    baseline and at the end of each 1-week treatment period. No abnormal 
    results in liver, kidney, leukocytes, erythrocytes, platelets, 
    electrolytes, or blood glucose were found in any of the patients at any 
    time. In this study vitamin E was used only in a combination product, 
    and each subject had a daily intake of 1,600 I.U. of vitamin E. 
    However, the combination product was only taken during 1 week of the 
    study. Therefore, the laboratory data do not provide any useful 
    information on the long-term effects of vitamin E.
        The second new clinical study (Ref. 5) was a four-period crossover 
    study in which each subject received 1,600 I.U. of vitamin E daily 
    (either singly or in a combination product) for 5 days during two of 
    the four treatment periods of the study. Although no laboratory tests 
    were performed, the subjects were asked to report any adverse reactions 
    at the end of each treatment period. Twenty-seven adverse reactions 
    were reported by 19 subjects out of 205 individuals completing all 
    phases of the study. Six of these adverse reactions were from subjects 
    who received vitamin E singly. Complaints included: Abdominal cramps, 
    nausea, loose bowels, and headache. The most commonly occurring 
    complaint was gastrointestinal disturbance (nausea, flatulence, or 
    diarrhea) of a transient nature. These reactions are consistent with 
    those previously reported in other studies; however, the investigators 
    considered the reactions as not related or probably not related to the 
    study drug.
        In the third new clinical study (Ref. 6), vitamin E (1,600 I.U. 
    daily for 2 weeks) was compared with placebo, quinine sulfate, and a 
    combination of quinine sulfate and vitamin E for the treatment and/or 
    prevention of nocturnal leg muscle cramps. Details of this multicenter, 
    parallel-design study are described in section I.C., comment 12. 
    Vitamin E alone was administered to 137 subjects. Headache was the most 
    frequently reported adverse event, occurring in 23 subjects (16.8 
    percent). However, a similar rate of headache (21 percent) was reported 
    in subjects taking placebo. The investigators described only six of 
    these events as possibly related to the study medication. Other adverse 
    events described by the investigators as possibly related to vitamin E 
    included three of four reports of nausea, two of three reports of 
    myalgia, and one of three reports of local edema. Thus, daily doses of 
    1,600 I.U. of vitamin E were well tolerated in this study.
        Bendich and Machlin (Ref. 8) reviewed six double-blind studies 
    involving vitamin E at doses as high as 3,200 I.U. daily for up to 6 
    months. Very few adverse effects were noted, and no specific side 
    effect was consistently observed in all the studies. In one study, 202 
    college students received 600 I.U. of vitamin E or placebo daily for 28 
    days in a randomized, double-blind trial (Ref. 9). No effects on 
    prothrombin time, total blood leukocyte count, or serum creatine 
    phosphokinase activity were evident. In a randomized, double-blind, 
    placebo-controlled study, 30 healthy adults were given 800 I.U. of 
    vitamin E or placebo daily for 16 weeks. There were no significant 
    differences in effects on plasma lipids between the vitamin E and 
    placebo groups (Ref. 10). No side effects were observed in a double-
    blind, crossover study of 48 subjects who received 1,600 I.U. of 
    vitamin E or placebo daily for a period of 6 months (Ref. 11). There 
    were no reports of significant side effects, weakness, fatigue, or 
    thrombophlebitis in a double-blind, crossover study in which 2,000 I.U. 
    of vitamin E or placebo was given daily to 25 adult onset-diabetic 
    subjects for a period of 6 weeks (Ref. 12). Thyroid hormone levels were 
    found to be identical for both the treatment and placebo periods. Hale 
    et al. (Ref. 13) examined the incidence of various clinical disorders 
    and measured a number of laboratory variables in 369 subjects who used 
    vitamin E supplements and 1,861 subjects who did not. All subjects were 
    over age 65. Use of vitamin E appeared to have little influence on 
    clinical disorders or hematologic or biochemical parameters. Only the 
    serum glutamic oxaloacetic transaminase was higher in vitamin E users. 
    However, the values were still within the accepted normal range. There 
    were no significant differences between users and nonusers in the 
    prevalence of hypertension, vaginal bleeding, frequent headache, 
    dizziness, recurrent diarrhea, diabetus mellitus, lightheaded-ness, or 
    thyroid disorders.
        Roberts (Ref. 7) raised concerns about an increased incidence of 
    thrombophlebitis associated with excessive vitamin E intake. In over 10 
    years of practice, Roberts encountered more than 80 patients with 
    problems that he attributed to self-medication with high doses of 
    vitamin E (greater than 800 I.U. daily). He suggested that vitamin E 
    may encourage thrombosis in patients with a predisposing condition. 
    Symptoms of thrombophlebitis were said to have abated upon cessation of 
    vitamin E therapy. Conventional treatment for thrombophlebitis (e.g., 
    bed rest, local heat) was administered along with the discontinuation 
    of vitamin E therapy. Thus, it is difficult to assess which action was 
    responsible for the improvement. In addition, no controlled studies or 
    concurrent references were included in support of his conclusions.
        Fitzgerald and Brash (Ref. 14) stated that vitamin E at 1,600 I.U. 
    a day in humans decreases platelet thromboxane production which could 
    consequently reduce the potential for thrombosis formation. In 
    addition, they noted that associations between thrombophlebitis and 
    vitamin E use have not been reported by other authors.
        Several authors (Refs. 2, 9, and 15) have reported that oral intake 
    of high doses of vitamin E has not produced blood coagulation 
    abnormalities in normal humans. However, in individuals deficient in 
    vitamin K (caused by malabsorption, diet, or anticoagulant therapy), 
    large doses of vitamin E can exacerbate coagulation defects. Therefore, 
    high levels of supplemental vitamin E may be contraindicated in such 
    conditions (Ref. 8).
        Based on the discussion above, the agency concludes that sufficient 
    evidence exists to support the safety of vitamin E at the daily doses 
    that have been commonly used for the treatment and/or prevention of 
    nocturnal leg muscle cramps.
    
    References
    
        (1) Salkeld, R. M., ``Safety and Tolerance of High-Dose Vitamin 
    E Administration in Man: A Review of the Literature,'' draft of 
    unpublished data in Comment No. C124, Docket No. 77N-0094, Dockets 
    Management Branch.
        (2) Farrell, P. M., and J. G. Bieri, ``Megavitamin E 
    Supplementation in Man,'' The American Journal of Clinical 
    Nutrition, 28:1381-1386, 1975.
        (3) Hathcock, J., ``Vitamin Safety: A Current Appraisal,'' in 
    ``Vitamin Issues,'' Vol. V, No. 1, published by Vitamin Nutrition 
    Information Service, in Comment No. C122, Docket No. 77N-0094, 
    Dockets Management Branch.
        (4) Biodesign GmbH, ``Clinical Evaluation of Q-VELR in Patients 
    with Nocturnal Leg Muscle Cramps,'' draft of an unpublished paper in 
    Comment No. SUP00031, Docket No. 77N-0094, Dockets Management 
    Branch.
        (5) Leo Winter Associates, Inc., ``Final Medical Report and Data 
    Summary Analysis and Final Statistical Report on Double-Blind 
    Randomized Crossover Study of Q-VELR Versus Quinine Sulfate Versus 
    Vitamin E Versus Placebo in the Treatment of Nocturnal Leg Muscle 
    Cramps (No. 1285-5082),'' draft of an unpublished paper in Comment 
    No. SUP00031, Docket No. 77N-0094, Dockets Management Branch.
        (6) Draft of an unpublished study entitled ``A Short-Term, 
    Randomized, Double-Blind, Parallel Study of Q-Vel vs. Quinine 
    Sulfate vs. Vitamin E vs. Placebo in the Prevention and Treatment of 
    Nocturnal Leg Cramps,'' Comment No. SUP00033, Docket No. 77N-0094, 
    Dockets Management Branch.
        (7) Roberts, H. J., ``Perspective On Vitamin E as Therapy,'' 
    Journal of the American Medical Association, 246(2):129-131, 1981.
        (8) Bendich, A., and L. J. Machlin, ``Safety of Oral Intake of 
    Vitamin E,'' The American Journal of Clinical Nutrition, 48:612-619, 
    1988.
        (9) Tsai, A. C. et al., ``Study on the Effect of Megavitamin E 
    Supplementation in Man, ''The American Journal of Clinical 
    Nutrition, 31(5):831-837, 1978.
        (10) Stampfer, M. J. et al., ``Effect of Vitamin E on Lipids,'' 
    American Journal of Clinical Pathology, 79(6):714-716, 1983.
        (11) Gillilan, R. E., B. Mondell, and J. R. Warbasse, 
    ``Quantitative Evaluation of Vitamin E in the Treatment of Angina 
    Pectoris,''American Heart Journal, 93(4):444-449, 1977.
        (12) Bierenbaum, M. L. et al., ``The Effect of Supplemental 
    Vitamin E on Serum Parameters in Diabetics, Post Coronary and Normal 
    Subjects,'' Nutrition Report International, 31(6):1171-1180, 1985.
        (13) Hale, W. E. et al., ``Vitamin E Effect on Symptoms and 
    Laboratory Values in the Elderly,'' Journal of the American Dietetic 
    Association, 86(5):625-629, 1986.
        (14) Fitzgerald, G. A., and A. R. Brash, ``Endogenous 
    Prostacyclin and Thromboxane Biosynthesis During Chronic Vitamin E 
    Therapy in Men,'' Annals of the New York Academy of the Sciences, 
    393:209-211, 1982.
        (15) Corrigan, J. J., ``The Effect of Vitamin E on Warfarin-
    Induced Vitamin K Deficiency,'' Annals of the New York Academy of 
    Sciences, 393:361-368, 1982.
    
    C. Comments on the Effectiveness of Nocturnal Leg Muscle Cramp 
    Ingredients
    
        12. One comment disagreed with the agency's Category III 
    classification of quinine sulfate for the treatment and/or prevention 
    of nocturnal leg muscle cramps on the basis of a lack of adequate 
    clinical data demonstrating the effectiveness of quinine sulfate for 
    this indication (50 FR 46588 at 46590). The comment contended that 
    there is sufficient evidence of quinine's effectiveness for this 
    indication at present to warrant classifying it in Category I. The 
    comment subsequently submitted the results of two clinical studies 
    (Refs. 1 and 2) comparing quinine sulfate, vitamin E, a combination 
    product containing quinine sulfate and vitamin E, and placebo for the 
    treatment and prevention of nocturnal leg muscle cramps to support the 
    effectiveness of the individual ingredients (quinine sulfate and 
    vitamin E) as well as the combination of these ingredients for this 
    indication. Another comment provided the results of three clinical 
    studies (Refs. 3, 4, and 5) that it felt addressed the effectiveness 
    issues raised by the agency in the tentative final monograph (50 FR 
    46590). This comment requested an oral hearing if the submitted data 
    were not found adequate to upgrade quinine sulfate to Category I. In 
    addition, in response to a citizen petition, one comment included the 
    results of a clinical study intended to demonstrate the efficacy of a 
    combination product containing quinine sulfate and vitamin E (Ref. 6).
        In the tentative final monograph, the agency concluded, on the 
    basis of its review of the new data submitted and the studies and 
    information discussed by the Internal Analgesic and Miscellaneous 
    Internal Panels, that quinine sulfate for use in OTC drug products for 
    the treatment and/or prevention of nocturnal leg muscle cramps should 
    be classified in Category III. The agency stated that adequate clinical 
    data are necessary to support the reclassification of quinine from 
    Category III to Category I and that any such studies should address the 
    following safety and effectiveness issues (50 FR 46588 at 46590):
        (1) Is quinine effective in treating and/or preventing nocturnal 
    leg muscle cramps in low daily doses (e.g., 200 to 325 mg) over short 
    periods of time (e.g., 7 days or less)?
        (2) If short-term quinine treatment with low doses is not 
    significantly effective in reducing recurrent episodes of nocturnal leg 
    muscle cramps, must such medication be taken over extended periods of 
    time to obtain relief? If yes, how long a period of time?
        (3) What adverse effects are experienced by subjects exposed to 
    effective doses of quinine over an effective course of therapy?
        The agency has reviewed the additional clinical data that have been 
    submitted and determined that they are not adequate to support the 
    reclassification of quinine sulfate to Category I for this use. Three 
    studies (Refs. 3, 4, and 5) compared quinine to placebo. In one study 
    (Ref. 3), 75 subjects were enrolled in a double-blind, randomized, 
    placebo-controlled, crossover study that was conducted over a 10-week 
    period in five 2-week intervals. Subjects with a history of at least 
    two cramps per week for at least 3 months were included in this study 
    and randomized to one of two treatment groups (group I or II). The 
    initial 2-week period was a baseline period and patients who failed to 
    have at least two cramps per week were dropped from the study. Subjects 
    who had a sufficient number of cramps during the baseline period were 
    either given the placebo (group I) or 325 mg of quinine sulfate per 
    night (group II) for a period of 2 weeks. No treatment was given for 
    the next 2-week period and in the fourth 2-week period subjects crossed 
    over to the alternate treatment. A final 2-week period of no treatment 
    followed. Subjects were issued weekly case report forms upon which they 
    were instructed to record the number of cramps experienced per night, 
    the time of the cramp, and the severity of the cramp. Subjects were 
    also asked to rate the effectiveness of the medication just completed 
    at weeks 4 and 8 of the study. According to the protocol, subjects were 
    assigned to the treatment sequence, on the basis of a predetermined 
    randomization schedule, prior to entering the baseline period. 
    Therefore, the removal of subjects from the study in the first 2 weeks 
    for not having enough events may have biased the study.
        In the statistical analysis of the study data, three efficacy 
    variables were evaluated: The mean frequency of leg cramps (per night), 
    the mean severity of leg cramps per night, and the total number of 
    nights per week that leg cramps occurred. The last variable (total 
    number of nights per week that leg cramps occurred) appears to be 
    constructed from the primary data because no such variable is listed on 
    the weekly case report forms from which these variables are derived. 
    The subjects' overall assessment of the effectiveness of the drug was 
    collected but not analyzed.
        Sixty-two of the 75 subjects enrolled in the study were included in 
    the data analysis. Of the 13 subjects found to be unevaluable, 8 
    withdrew from the study on their own accord. No specific reasons for 
    these withdrawals were given, but it is stated in the study report that 
    they were unrelated to the treatment. The remaining five subjects were 
    dropped for various medical reasons and noncompliance with the 
    protocol. No ``intent-to-treat'' analysis was performed.
        A number of analyses were carried out. Two of the analyses treated 
    the unblinded baseline and washout periods as if they were treatment 
    periods. This type of analysis is incorrect for a crossover trial. The 
    relevant comparisons that should be made are between the treatments in 
    the double-blind periods, possibly with adjustments for baseline, 
    provided there are no major changes in baseline values for each period.
        When Patel's joint test for equal carryover and equal pretreatment 
    severity (Ref. 7) is applied to the data, however, significant 
    differences are seen in pretreatment severity before the second period. 
    Analysis of the second period is thus compromised; therefore, analysis 
    should be limited to the first treatment period (weeks 3 and 4). This 
    comparison does not show a significant advantage for quinine sulfate 
    over placebo for any of the effectiveness variables.
        Another clinical study (Ref. 4) used the same study design as the 
    study discussed above except that the dose of quinine sulfate was 260 
    mg/night, not the 325 mg/night used in the first study. In addition, 
    five efficacy variables were analyzed: Frequency, severity, and 
    duration of leg cramps, and induction and quality of sleep.
        Although the predetermined randomization chart submitted for this 
    study provided for enrollment of 74 subjects, 84 subjects entered the 
    study. No explanation for entry of the additional 10 subjects was 
    provided. As in the first study, randomization to treatment sequence 
    occurred at the time of entry into the baseline period; thus, 
    subsequent removal of subjects prior to the first double-blind 
    treatment period may also have introduced bias into this study. Of the 
    84 subjects entered at baseline, 69 (34 assigned to group I and 35 to 
    group II) entered the double- blind treatment phase.
        The study concluded that significant differences at the 5-percent 
    level exist between quinine sulfate and placebo for three of the five 
    variables: Frequency of cramps, induction of sleep, and quality of 
    sleep. However, no documentation of any statistical analysis supporting 
    these claims was provided.
        The statistical report that accompanied the study addressed the 
    question of comparing the effectiveness of quinine sulfate and placebo 
    with a multivariate analysis of covariance which compared the vector of 
    efficacy variables over four observation periods (two treatment periods 
    plus two washout periods with the initial baseline value as a 
    covariate). The conclusion of the analysis was that the treatment 
    effect was not significant (p = 0.106). Univariate analyses of 
    covariance comparing these four observation periods were referred to in 
    the statistical report, but no p-values for treatment effect were 
    provided (although a significant order by treatment interaction was 
    reported). Also included in the statistical analyses of the study were 
    comparisons of the four observation periods separately by sequence 
    (quinine sulfate-placebo and placebo-quinine sulfate), which included 
    baseline-adjusted means and comparisons between periods using Duncan's 
    Multiple Range Test. These comparisons showed that significant 
    differences were demonstrated between quinine and placebo only for the 
    placebo-quinine sulfate sequence (group II), and only for three 
    variables: Frequency of cramps, quality of sleep, and induction of 
    sleep. However, the adjusted means for the quinine sulfate-placebo 
    sequence (group I) favored placebo over quinine sulfate for all five 
    efficacy variables. In addition, as for the first study, the 
    appropriate statistical analysis for this type of study was not done. 
    The hypothesis of equal carryover effect was not tested and not 
    rejected before any of the other statistical tests for treatment effect 
    were performed. The results of this study are not adequate to support 
    the effectiveness of quinine sulfate for the treatment and/or 
    prevention of nocturnal leg muscle cramps.
        The study by Jones and Castleden (Ref. 5) also does not provide 
    adequate evidence of quinine sulfate's effectiveness for this 
    indication. The study was a double-blind crossover study of nine 
    patients with four 2-week periods of observation (a run-in period and a 
    washout period in addition to two treatment periods of placebo or 
    quinine sulfate 300 mg/day). The same five efficacy values as in the 
    second study above were evaluated: Frequency, severity, and duration of 
    leg cramps, and induction and quality of sleep. No raw data were 
    included to substantiate any of the statistical claims made by the 
    authors; nor was a protocol included in the article.
        Of the five primary efficacy variables, only severity of cramps was 
    claimed to show a significant difference between quinine sulfate and 
    placebo (p < 0.025),="" although="" an="" analysis="" of="" frequency="" of="" cramps="" after="" 2="" a.m.="" was="" also="" claimed="" to="" be="" significant="" (p="">< 0.025).="" there="" was="" no="" indication="" that="" the="" time="" period="" after="" 2="" a.m.="" was="" identified="" in="" the="" protocol="" as="" defining="" a="" primary="" endpoint;="" thus,="" this="" is="" assumed="" to="" be="" a="" post-hoc="" analysis="" done="" after="" reviewing="" the="" data.="" in="" general,="" the="" isolated="" severity="" finding="" is="" not="" convincing="" on="" its="" face.="" in="" addition,="" the="" published="" article="" did="" not="" provide="" sufficient="" information="" to="" permit="" an="" independent="" analysis="" of="" the="" data.="" for="" these="" reasons,="" the="" study="" does="" not="" provide="" evidence="" that="" quinine="" sulfate="" is="" an="" effective="" treatment="" for="" nocturnal="" leg="" muscle="" cramps.="" three="" studies="" (refs.="" 1,="" 2,="" and="" 6)="" were="" submitted="" to="" support="" the="" effectiveness="" of="" quinine="" sulfate="" and="" vitamin="" e="" individually="" and="" in="" combination="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" the="" freiburg="" study="" (ref.="" 1)="" was="" a="" 5-week,="" double-blind,="" randomized,="" crossover="" study="" in="" 24="" subjects.="" all="" subjects="" received="" placebo="" during="" week="" 1="" (baseline),="" week="" 3,="" and="" week="" 5.="" subjects="" in="" group="" i="" received="" quinine="" in="" week="" 2="" and="" the="" combination="" of="" quinine="" and="" vitamin="" e="" in="" week="" 4,="" while="" subjects="" in="" group="" ii="" were="" given="" the="" combination="" product="" in="" week="" 2="" and="" quinine="" in="" week="" 4.="" a="" statistically="" significant="" difference="" in="" frequency="" of="" attacks="" between="" the="" combination="" product="" and="" quinine="" sulfate="" was="" reported,="" but="" no="" difference="" in="" duration="" or="" severity="" of="" attack="" was="" found="" between="" these="" two="" active="" treatments.="" the="" report="" described="" an="" ``obvious''="" improvement="" in="" frequency,="" duration,="" and="" severity="" of="" attacks="" between="" the="" placebo="" periods="" and="" both="" active="" treatments,="" but="" no="" statistical="" evidence="" or="" analysis="" to="" support="" this="" conclusion="" was="" provided.="" moreover,="" the="" comparison="" of="" treatments="" and="" placebo="" did="" not="" involve="" randomized="" patient="" groups,="" nor="" was="" it="" blinded.="" only="" the="" portion="" of="" the="" study="" comparing="" the="" combination="" product="" versus="" quinine="" was="" a="" randomized,="" double-blind="" trial.="" the="" study="" report="" did="" not="" include="" the="" study="" protocol,="" details="" of="" the="" statistical="" analysis="" conducted,="" or="" individual="" subject="" data.="" the="" model="" described="" in="" the="" summary="" of="" the="" data="" analysis="" did="" not="" properly="" separate="" carryover="" effect="" from="" treatment="" effect.="" the="" study="" provides="" no="" evidence="" from="" a="" controlled="" trial="" that="" quinine="" is="" effective="" for="" nocturnal="" leg="" muscle="" cramps.="" the="" other="" study="" (ref.="" 2)="" also="" employed="" a="" complicated="" randomized,="" four-period,="" crossover="" design.="" there="" were="" 205="" subjects="" randomly="" assigned="" to="" one="" of="" four="" treatment="" groups:="" quinine="" sulfate="" 260="" mg/day,="" vitamin="" e="" 1,600="" i.u./day,="" a="" combination="" of="" quinine="" and="" vitamin="" e,="" or="" placebo.="" the="" combination="" of="" quinine="" and="" vitamin="" e="" was="" reported="" as="" being="" statistically="" superior="" to="" both="" its="" components="" and="" placebo="" for="" six="" variables:="" effect="" of="" cramps="" on="" falling="" asleep,="" nighttime="" awakening="" due="" to="" cramps,="" number="" of="" cramps,="" severity="" of="" cramps,="" subject="" global="" evaluation,="" and="" difficulty="" falling="" asleep="" due="" to="" cramps.="" the="" study="" also="" reported="" statistically="" significant="" positive="" findings="" on="" quinine="" sulfate="" versus="" placebo="" for="" the="" first="" five="" of="" these="" six="" variables.="" as="" in="" the="" freiburg="" study,="" the="" model="" used="" in="" the="" statistical="" analysis="" does="" not="" properly="" separate="" the="" carryover="" effect="" from="" the="" treatment="" effect.="" neither="" the="" data="" listings="" nor="" the="" results="" by="" period="" were="" provided.="" therefore,="" the="" agency="" was="" unable="" to="" independently="" analyze="" the="" results="" of="" this="" study="" or="" to="" rely="" on="" the="" analysis="" provided="" as="" evidence="" that="" the="" reported="" results="" were="" attributable="" to="" drug="" treatment.="" the="" third="" clinical="" study="" compared="" quinine="" sulfate,="" vitamin="" e,="" and="" a="" combination="" of="" quinine="" sulfate="" and="" vitamin="" e="" to="" placebo,="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" (ref.="" 6).="" this="" study="" was="" a="" multicenter,="" randomized,="" block="" parallel-design="" with="" a="" single-blind,="" placebo,="" run-in="" period,="" followed="" by="" a="" 2-week,="" double-="" blind,="" randomized,="" treatment="" phase.="" subjects="" who="" had="" at="" least="" one="" leg="" cramp="" per="" night="" for="" a="" minimum="" of="" 3="" nights="" during="" the="" single-blind="" placebo="" week,="" and="" met="" all="" other="" selection="" criteria,="" were="" randomly="" assigned="" to="" one="" of="" the="" four="" double-blind="" treatment="" groups.="" capsules,="" identical="" in="" appearance,="" contained="" either="" placebo,="" quinine="" sulfate="" 64.8="" mg,="" vitamin="" e="" 400="" i.u.,="" or="" a="" combination="" of="" quinine="" sulfate="" 64.8="" mg="" and="" vitamin="" e="" 400="" i.u.="" subjects="" were="" instructed="" to="" take="" two="" capsules="" following="" their="" evening="" meal,="" and="" two="" capsules="" before="" bedtime,="" which="" provided="" daily="" doses="" of="" 259.2="" mg="" of="" quinine="" sulfate,="" 1,600="" i.u.="" of="" vitamin="" e,="" or="" the="" combination="" thereof.="" efficacy="" endpoints="" identified="" in="" the="" protocol="" were:="" (1)="" number="" of="" episodes="" of="" nocturnal="" leg="" cramps="" per="" week,="" (2)="" sleep="" disturbance="" due="" to="" nocturnal="" leg="" cramps,="" (3)="" severity="" of="" nocturnal="" leg="" cramps,="" and="" (4)="" duration="" of="" nocturnal="" leg="" cramps.="" however,="" none="" of="" the="" parameters="" was="" designated="" as="" a="" primary="" efficacy="" variable="" in="" the="" protocol.="" the="" protocol="" specified="" that="" efficacy="" would="" be="" analyzed="" by="" analysis="" of="" variance="" with="" repeated="" measures="" test,="" as="" well="" as="" other="" methods="" deemed="" appropriate.="" on="" the="" basis="" of="" an="" estimated="" 30="" percent="" difference="" between="" the="" combination="" product="" and="" its="" components,="" assuming="" an="" alpha="" of="" 0.05="" and="" statistical="" power="" of="" 70="" percent,="" a="" sample="" size="" of="" 972="" evaluable="" subjects="" was="" planned="" (243="" subjects/group).="" enrollment="" was="" suspended,="" however,="" and="" the="" data="" were="" analyzed="" after="" 498="" evaluable="" subjects="" (51="" percent)="" completed="" the="" study.="" subjects="" were="" approximately="" evenly="" distributed="" among="" treatment="" groups.="" in="" the="" final="" report,="" results="" were="" separately="" analyzed="" for="" weeks="" 1="" and="" 2="" of="" the="" double-blind="" treatment.="" the="" change="" from="" baseline="" scores="" obtained="" during="" the="" single-blind,="" placebo="" week="" was="" analyzed="" on="" seven="" variables="" for="" each="" of="" the="" treatment="" groups="" at="" days="" 7="" and="" 14="" using="" a="" two-way="" analysis="" of="" variance="" test="" with="" terms="" for="" treatment,="" center,="" and="" treatment="" by="" center="" interaction.="" the="" data="" were="" not="" analyzed="" using="" the="" analysis="" of="" variance="" with="" repeated="" measures="" test,="" as="" prospectively="" stated="" in="" the="" protocol.="" the="" variables="" were:="" (1)="" number="" of="" nights="" per="" week="" with="" leg="" cramps,="" (2)="" average="" number="" of="" leg="" cramps="" per="" night,="" (3)="" average="" severity="" of="" leg="" cramps="" per="" night,="" (4)="" average="" duration="" of="" leg="" cramps="" per="" night,="" (5)="" average="" number="" of="" leg="" cramps="" per="" night="" with="" sleeping="" difficulty,="" (6)="" average="" degree="" of="" difficulty="" getting="" to="" sleep="" per="" night,="" and="" (7)="" average="" number="" of="" nights="" per="" week="" awakened="" by="" leg="" cramps.="" the="" placebo="" group="" was="" compared="" with="" the="" remaining="" treatment="" groups="" with="" the="" least-significant-difference="" test="" using="" error="" mean="" square="" from="" the="" analysis="" of="" variance="" table.="" within="" each="" treatment="" group,="" the="" amount="" of="" change="" from="" baseline="" for="" each="" efficacy="" parameter="" was="" compared="" for="" each="" double-blind="" treatment="" week="" using="" the="" wilcoxon="" sign="" rank="" test.="" p-values="" of="" 0.05="" or="" less="" were="" considered="" statistically="" significant.="" twelve="" centers="" initially="" participated="" in="" the="" study.="" three="" centers="" were="" terminated="" because="" of="" low="" enrollment="" (less="" than="" four="" evaluable="" subjects="" in="" at="" least="" one="" treatment="" group).="" these="" low="" enrollment="" centers="" were="" combined="" in="" the="" analysis.="" in="" the="" final="" report,="" the="" number="" of="" nights="" per="" week="" with="" leg="" cramps="" was="" declared="" the="" primary="" efficacy="" variable.="" during="" the="" baseline="" period,="" a="" mean="" of="" approximately="" 5="" nights="" per="" week="" with="" leg="" cramps="" was="" recorded="" in="" all="" groups="" (placebo="" 4.72,="" combination="" 4.95,="" quinine="" sulfate="" 5.04,="" vitamin="" e="" 4.98).="" all="" groups="" improved="" during="" week="" 1="" with="" a="" reduction="" in="" frequency="" to="" approximately="" 4="" nights="" per="" week="" with="" cramps="" (placebo="" 4.04,="" combination="" 3.73,="" quinine="" sulfate="" 3.53,="" vitamin="" e="" 3.97).="" the="" greatest="" reductions="" were="" in="" subjects="" given="" quinine="" sulfate="" and="" the="" combination="" product="" and="" the="" difference="" in="" week="" 1="" was="" found="" to="" be="" statistically="" significant="" compared="" to="" placebo="" for="" these="" treatment="" groups="" (p="" less="" than="" or="" equal="" to="" 0.04).="" statistically="" significant="" differences="" between="" quinine="" sulfate="" and="" placebo="" were="" reported="" in="" the="" first="" week="" of="" the="" study="" for="" four="" of="" the="" six="" remaining="" efficacy="" variables="" declared="" to="" be="" secondary="" parameters="" in="" the="" final="" report.="" quinine="" was="" reported="" to="" be="" significantly="" better="" than="" placebo="" in="" reducing="" the="" average="" number="" of="" leg="" cramps="" per="" night,="" average="" severity="" of="" leg="" cramps="" per="" night,="" average="" duration="" of="" leg="" cramps="" per="" night,="" and="" average="" number="" of="" nights="" per="" week="" with="" sleeping="" difficulty.="" no="" statistically="" significant="" differences="" between="" any="" of="" the="" treatment="" groups="" for="" any="" variable="" were="" reported="" for="" the="" second="" week="" of="" the="" study.="" the="" comment="" concluded="" that="" quinine="" sulfate,="" alone="" and="" in="" combination="" with="" vitamin="" e,="" at="" a="" daily="" dose="" of="" approximately="" 260="" mg="" was="" safe="" and="" effective="" in="" the="" short="" term="" (1-week)="" treatment="" of="" nocturnal="" leg="" muscle="" cramps.="" the="" agency="" finds="" that="" there="" were="" a="" number="" of="" flaws="" in="" the="" analysis="" of="" this="" study.="" first,="" the="" primary="" endpoint="" (number="" of="" nights="" per="" week="" with="" leg="" cramps)="" appears="" to="" have="" been="" arbitrarily="" chosen="" after="" the="" study="" was="" completed.="" none="" of="" the="" efficacy="" variables="" was="" declared="" the="" primary="" endpoint="" in="" the="" protocol.="" second,="" the="" study="" was="" of="" 2="" week's="" duration,="" and="" there="" was="" no="" provision="" in="" the="" protocol="" for="" separate="" evaluation="" of="" the="" data="" from="" week="" 1="" and="" week="" 2.="" thus,="" there="" is="" no="" basis="" for="" the="" decision="" to="" analyze="" week="" 1="" and="" week="" 2="" separately="" in="" the="" absence="" of="" such="" an="" analysis="" declared="" prospectively="" in="" the="" protocol.="" in="" fact,="" an="" analysis="" of="" both="" weeks="" together="" (see="" below)="" does="" not="" show="" a="" significant="" benefit="" of="" quinine.="" third,="" an="" adjustment="" for="" multiple="" comparisons="" should="" have="" been="" included="" in="" the="" data="" analysis.="" given="" seven="" variables,="" two="" active="" treatments,="" and="" at="" least="" three="" time="" points="" at="" which="" data="" could="" be="" analyzed="" (first="" week,="" second="" week,="" both="" weeks),="" the="" nominally="" significant="" differences="" between="" treatments="" at="" the="" end="" of="" week="" one="" would="" not="" be="" expected="" to="" retain="" statistical="" significance="" if="" an="" adjustment="" for="" multiple="" comparisons="" were="" included="" in="" the="" analysis.="" even="" considering="" the="" retrospectively="" identified="" primary="" endpoint,="" a="" correction="" for="" three="" ``looks''="" (week="" 1,="" week="" 2,="" and="" together)="" would="" at="" least="" double="" the="" nominal="" p-value.="" even="" without="" correction="" for="" multiplicity,="" the="" results="" do="" not="" support="" the="" conclusion="" that="" quinine="" sulfate="" and="" vitamin="" e,="" alone="" or="" in="" combination,="" are="" effective="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" first,="" week="" 2="" results="" fail="" to="" replicate="" the="" results="" of="" week="" 1.="" no="" differences="" between="" any="" of="" the="" treatment="" groups="" for="" any="" parameter="" were="" found="" at="" the="" end="" of="" week="" 2,="" nor="" was="" the="" investigators'="" global="" assessment,="" conducted="" at="" the="" end="" of="" the="" 2-week="" double-blind="" period,="" able="" to="" differentiate="" between="" treatments.="" second,="" a="" significant="" treatment="" by="" center="" interaction="" was="" found="" for="" the="" reported="" superiority="" of="" quinine="" sulfate="" over="" placebo="" in="" week="" 1="" in="" reducing="" the="" number="" of="" nights="" per="" week="" with="" leg="" cramps.="" the="" result="" was="" driven="" by="" two="" of="" nine="" centers.="" in="" one="" of="" these="" centers,="" the="" combination="" product="" was="" indistinguishable="" from="" placebo,="" and="" in="" the="" other,="" the="" superiority="" of="" placebo="" over="" the="" combination="" neared="" statistical="" significance="" (p="0.10)." thus,="" in="" the="" two="" clinics="" responsible="" for="" the="" favorable="" week="" 1="" results="" of="" treatment="" with="" quinine,="" there="" was="" a="" failure="" to="" replicate="" the="" result="" reported="" with="" quinine="" sulfate="" alone.="" vitamin="" e="" was="" ineffective="" in="" all="" parameters="" measured="" throughout="" the="" study.="" the="" four="" retrospectively-declared="" secondary="" endpoints="" for="" which="" statistically="" significant="" reductions="" were="" reported="" in="" week="" 1="" in="" the="" quinine="" sulfate="" group="" compared="" to="" placebo="" were:="" (1)="" the="" number="" of="" cramps="" per="" night,="" (2)="" the="" number="" of="" nights="" with="" sleeping="" difficulty,="" (3)="" the="" severity="" of="" the="" cramps,="" and="" (4)="" the="" duration="" of="" the="" cramps.="" although="" a="" consistent="" benefit="" on="" these="" endpoints="" would="" render="" a="" finding="" on="" the="" primary="" endpoint="" more="" persuasive,="" as="" with="" the="" primary="" efficacy="" endpoint,="" none="" of="" the="" differences="" between="" active="" treatment="" and="" placebo="" persisted="" through="" to="" the="" end="" of="" week="" 2.="" for="" the="" reasons="" discussed="" above,="" the="" post="" hoc,="" week-1="" analysis="" of="" these="" endpoints="" fails="" to="" provide="" convincing="" evidence="" to="" support="" the="" efficacy="" of="" quinine="" sulfate="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" two="" additional="" analyses="" of="" the="" results="" of="" the="" study="" were="" submitted="" (refs.="" 8="" and="" 9).="" the="" first="" (ref.="" 8)="" was="" an="" analysis="" of="" the="" number="" of="" leg="" cramps="" per="" day="" for="" each="" day="" of="" the="" study.="" this="" analysis="" showed="" occasional="" days="" in="" which="" quinine="" was="" superior="" to="" placebo,="" but="" is="" on="" the="" whole="" not="" helpful.="" given="" an="" entry="" cramp="" rate="" of="" one="" cramp="" episode="" per="" night="" for="" at="" least="" 3="" nights="" per="" week,="" significant="" differences="" in="" any="" endpoint="" would="" not="" be="" expected="" on="" a="" day-by-day="" (i.e.,="" noncumulative)="" evaluation.="" the="" second="" analysis="" (ref.="" 9)="" was="" of="" the="" total="" cramp="" rate="" (mean="" number="" of="" cramps="" per="" day="" over="" the="" course="" of="" the="" entire="" study="" period)="" for="" both="" the="" evaluable="" subset="" of="" subjects="" and="" the="" intent-to-treat="" population.="" two="" analyses="" were="" performed="" on="" each="" group.="" in="" one="" analysis,="" only="" those="" subjects="" who="" completed="" the="" study="" with="" at="" least="" 14="" days="" of="" treatment="" (the="" completer="" analysis)="" were="" analyzed,="" while="" the="" other="" analysis="" involved="" the="" results="" from="" all="" subjects="" with="" efficacy="" observations="" (the="" endpoint="" analysis)="" for="" the="" quinine="" sulfate="" and="" placebo="" treatment="" groups.="" in="" the="" endpoint="" analyses,="" where="" less="" than="" 14="" days="" of="" treatment="" was="" completed,="" leg="" cramps="" for="" the="" observed="" number="" of="" days="" were="" calculated,="" and="" the="" mean="" was="" carried="" forward="" to="" 14="" days.="" none="" of="" the="" four="" analyses="" revealed="" statistically="" significant="" reductions="" in="" the="" mean="" number="" of="" leg="" cramps="" experienced="" during="" 14="" days="" of="" treatment="" in="" the="" quinine-treated="" subjects="" compared="" with="" placebo="" subjects.="" the="" endpoint="" analysis="" for="" evaluable="" patients="" approached="" statistical="" significance="" for="" quinine="" sulfate="" (p="0.06)," but="" the="" results="" of="" the="" completer="" analysis="" for="" evaluable="" subjects="" and="" both="" intent-to-treat="" analyses="" were="" clearly="" negative.="" the="" total="" cramp="" rate="" over="" the="" entire="" study="" is="" the="" most="" straightforward="" effectiveness="" measure;="" it="" did="" not="" show="" a="" drug="" effect="" on="" cramps.="" while="" the="" favorable="" trend="" on="" one="" analysis="" could="" suggest="" activity,="" the="" study="" was="" already="" of="" very="" substantial="" size="" and="" should="" have="" been="" able="" to="" detect="" a="" clinically="" meaningful="" response.="" this="" study,="" therefore,="" does="" not="" provide="" evidence="" of="" efficacy="" of="" quinine="" sulfate,="" vitamin="" e,="" or="" the="" combination="" thereof="" in="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" based="" on="" the="" above="" discussion,="" the="" agency="" concludes="" that="" the="" submitted="" data="" are="" inadequate="" to="" establish="" the="" effectiveness="" of="" quinine="" sulfate="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" further,="" the="" agency="" concludes="" that="" the="" submitted="" data="" do="" not="" adequately="" address="" the="" safety="" and="" effectiveness="" issues="" raised="" by="" the="" agency="" in="" the="" tentative="" final="" monograph="" (see="" discussion="" above).="" additional="" agency="" comments="" and="" evaluations="" of="" the="" data="" are="" on="" file="" in="" the="" dockets="" management="" branch="" (refs.="" 10,="" 11,="" and="" 12).="" the="" commissioner="" has="" determined="" that="" there="" are="" not="" reasonable="" grounds="" in="" support="" of="" a="" hearing="" and="" that="" a="" hearing="" on="" this="" issue="" is="" not="" warranted.="" six="" clinical="" trials="" have="" been="" submitted="" and="" have="" failed="" to="" establish="" the="" safety="" and="" efficacy="" of="" quinine="" sulfate="" in="" treating="" and/or="" preventing="" nocturnal="" leg="" muscle="" cramps.="" occasional="" significant="" differences="" favoring="" quinine="" were="" not="" replicated="" within="" or="" between="" studies.="" in="" two="" crossover="" design="" studies="" (refs.="" 3="" and="" 4),="" appropriate="" analyses="" revealed="" no="" significant="" differences="" between="" quinine="" sulfate="" and="" placebo.="" the="" results="" of="" a="" very="" large="" parallel-design,="" 2-week="" study="" showed="" no="" significant="" effect="" in="" an="" analysis="" of="" the="" 2-week="" data.="" in="" addition,="" deficiencies="" in="" the="" studies="" themselves="" render="" the="" reported="" results="" unreliable.="" each="" of="" these="" studies="" involved="" multiple="" endpoints,="" none="" of="" which="" was="" prospectively="" declared="" as="" the="" primary="" efficacy="" variable(s)="" in="" any="" of="" the="" studies.="" there="" was="" no="" attempt="" to="" correct="" significance="" levels="" for="" multiple="" endpoints.="" the="" design="" of="" one="" study="" did="" not="" permit="" the="" independent="" evaluation="" of="" the="" efficacy="" of="" quinine="" sulfate="" alone="" (ref.="" 1).="" in="" three="" crossover="" studies="" (refs.="" 2,="" 3,="" and="" 4),="" the="" treatment="" effect="" was="" confounded="" by="" potential="" carryover="" effect="" and="" baseline="" differences.="" the="" 2-week,="" parallel-design="" study="" (ref.="" 6)="" showed="" no="" effect="" overall="" for="" the="" entire="" treatment="" period,="" including="" the="" investigator's="" global="" assessment.="" only="" by="" considering="" the="" results="" of="" week="" 1="" separately,="" an="" unplanned="" analysis,="" was="" any="" significant="" difference="" between="" quinine="" and="" placebo="" found="" in="" this="" study,="" and="" this="" finding="" was="" confounded="" by="" a="" significant="" treatment-by-center="" interaction.="" for="" these="" reasons,="" the="" studies="" cannot="" be="" considered="" adequate="" and="" well-controlled="" clinical="" investigations="" as="" required="" under="" sec.="" 330.10(a)(4)(ii).="" the="" commissioner="" concludes="" that="" a="" hearing="" on="" this="" issue="" is="" not="" justified="" for="" the="" reasons="" stated="" above.="" references="" (1)="" biodesign="" gmbh,="" ``clinical="" evaluation="" of="" q-velr="" in="" patients="" with="" nocturnal="" leg="" muscle="" cramps,''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" sup00031,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (2)="" leo="" winter="" associates,="" inc.,="" ``final="" medical="" report="" and="" data="" summary="" analysis="" and="" final="" statistical="" report="" on="" double="" blind="" randomized="" crossover="" study="" of="" q-velr="" versus="" quinine="" sulfate="" versus="" vitamin="" e="" versus="" placebo="" in="" the="" treatment="" of="" nocturnal="" leg="" muscle="" cramps="" (no.="" 1285-5082),''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" sup00031,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (3)="" hays,="" r.,="" and="" j.="" j.="" goodman,="" ``clinical="" trial="" of="" the="" efficacy="" of="" quinine="" sulfate="" in="" the="" treatment="" of="" nocturnal="" leg="" muscle="" cramps,="" protocol="" 86-48,''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" c126,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (4)="" bottner,="" m.,="" ``clinical="" trial="" of="" the="" efficacy="" of="" quinine="" sulfate="" in="" the="" treatment="" of="" nocturnal="" leg="" muscle="" cramps,="" protocol="" 84-46,''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" c123,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (5)="" jones,="" k.,="" and="" c.="" m.="" castleden,="" ``a="" double-blind="" comparison="" of="" quinine="" sulphate="" and="" placebo="" in="" muscle="" cramps,''="" age="" and="" ageing,="" 12(2):155-158,="" 1983.="" (6)="" draft="" of="" an="" unpublished="" study="" entitled="" ``a="" short-term,="" randomized,="" double-blind,="" parallel="" study="" of="" q-vel="" vs.="" quinine="" sulfate="" vs.="" vitamin="" e="" vs.="" placebo="" in="" the="" prevention="" and="" treatment="" of="" nocturnal="" leg="" cramps,''="" comment="" no.="" sup00033,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (7)="" patel,="" h.,="" ``use="" of="" baseline="" measurements="" in="" the="" two-period="" cross-over="" design,''="" communications="" in="" statistics-theory="" and="" methods,="" 12(23):2693-2712,="" 1983.="" (8)="" comment="" no.="" c159,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (9)="" comment="" no.="" sup00041,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (10)="" letter="" from="" w.="" e.="" gilbertson,="" fda,="" to="" k.="" m.="" o'brien,="" scholl,="" inc.,="" coded="" let00059,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (11)="" letter="" from="" w.="" e.="" gilbertson,="" fda,="" to="" l.="" d.="" fantasia,="" ciba="" consumer="" pharmaceuticals,="" coded="" let00060,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (12)="" memorandum="" of="" telephone="" conversation="" between="" l.="" fantasia,="" ciba="" consumer="" pharmaceuticals,="" and="" l.="" geismar,="" fda,="" january="" 4,="" 1989,="" coded="" mt0009,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" 13.="" one="" comment="" disagreed="" with="" the="" agency's="" category="" iii="" classification="" of="" vitamin="" e="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" on="" the="" basis="" of="" a="" lack="" of="" adequate="" clinical="" data="" demonstrating="" the="" effectiveness="" of="" vitamin="" e="" for="" this="" indication="" (50="" fr="" 46588="" at="" 46591).="" the="" comment="" contended="" that="" there="" is="" sufficient="" evidence="" of="" vitamin="" e's="" effectiveness="" for="" this="" indication="" at="" present="" to="" warrant="" classifying="" it="" in="" category="" i.="" the="" comment="" subsequently="" submitted="" the="" results="" of="" two="" clinical="" studies="" (refs.="" 1="" and="" 2)="" comparing="" vitamin="" e,="" quinine="" sulfate,="" a="" combination="" product="" containing="" vitamin="" e="" and="" quinine="" sulfate,="" and="" placebo="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" to="" support="" the="" effectiveness="" of="" the="" individual="" ingredients="" (vitamin="" e="" and="" quinine="" sulfate)="" as="" well="" as="" the="" combination="" of="" these="" ingredients="" for="" this="" indication.="" in="" addition,="" in="" responding="" to="" a="" citizen="" petition,="" one="" comment="" included="" a="" clinical="" study="" comparing="" vitamin="" e,="" quinine="" sulfate,="" a="" combination="" product="" containing="" both="" ingredients,="" and="" placebo="" (ref.="" 3).="" in="" the="" tentative="" final="" monograph,="" the="" agency="" concluded="" that="" there="" was="" a="" lack="" of="" controlled="" studies="" demonstrating="" the="" effectiveness="" of="" vitamin="" e="" in="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" the="" agency="" also="" determined="" that="" a="" safe="" and="" effective="" otc="" dosage="" of="" vitamin="" e="" had="" not="" been="" established="" (50="" fr="" 46588="" at="" 46591).="" therefore,="" the="" agency="" classified="" vitamin="" e="" in="" category="" iii="" for="" this="" use.="" the="" agency="" has="" reviewed="" the="" additional="" clinical="" data="" that="" have="" been="" submitted="" and="" determined="" that="" they="" are="" not="" adequate="" to="" support="" the="" reclassification="" of="" vitamin="" e="" to="" category="" i="" for="" this="" use.="" in="" one="" double-blind,="" randomized,="" crossover="" study="" (ref.="" 1),="" a="" combination="" product="" containing="" 64.8="" mg="" quinine="" sulfate="" and="" 400="" i.u.="" of="" vitamin="" e="" in="" a="" lecithin="" base="" was="" compared="" to="" 64.8="" mg="" of="" quinine="" sulfate="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" in="" subjects="" with="" a="" history="" of="" nocturnal="" leg="" muscle="" cramps.="" subjects="" were="" randomized="" into="" two="" groups.="" all="" subjects="" took="" placebo="" during="" week="" 1="" and="" at="" the="" end="" of="" week="" 1="" only="" those="" subjects="" reporting="" at="" least="" three="" cramps="" per="" week="" were="" allowed="" to="" continue="" in="" the="" study.="" one="" group="" received="" the="" combination="" product="" during="" week="" 2="" and="" quinine="" sulfate="" during="" week="" 4,="" while="" for="" the="" other="" group="" this="" order="" was="" reversed.="" both="" groups="" also="" received="" placebo="" during="" weeks="" 3="" and="" 5.="" both="" quinine="" sulfate="" and="" the="" combination="" of="" quinine="" sulfate="" and="" vitamin="" e="" were="" reported="" to="" reduce="" the="" frequency="" of="" nocturnal="" leg="" muscle="" cramps="" in="" this="" study.="" a="" greater="" reduction="" in="" the="" frequency="" of="" these="" leg="" cramps="" was="" observed="" in="" subjects="" taking="" the="" combination="" product="" compared="" to="" subjects="" taking="" quinine="" alone.="" the="" difference="" was="" reported="" to="" be="" statistically="" significant="" using="" wilcoxon's="" signed-rank="" test.="" no="" significant="" differences="" were="" found="" between="" treatments="" for="" either="" duration="" or="" severity="" of="" attacks.="" however,="" as="" previously="" discussed="" (see="" section="" i.c.,="" comment="" 12),="" the="" study="" report="" did="" not="" include="" the="" study="" protocol,="" details="" of="" the="" statistical="" analysis,="" or="" individual="" subject="" data,="" and="" the="" analysis="" described="" does="" not="" properly="" separate="" carryover="" effect="" from="" treatment="" effect.="" therefore,="" it="" is="" not="" possible="" to="" conclude="" that="" either="" treatment="" used="" in="" this="" study="" was="" effective="" for="" this="" indication.="" the="" second="" clinical="" study="" (ref.="" 2)="" was="" a="" double-blind,="" randomized,="" crossover="" study="" conducted="" at="" two="" sites="" and="" involved="" subjects="" with="" at="" least="" a="" 3-month="" history="" of="" at="" least="" two="" significant="" nocturnal="" leg="" muscle="" cramps="" per="" week.="" the="" subjects="" did="" not="" receive="" any="" drug="" for="" the="" first="" 1-week="" run-in="" period,="" then="" received="" four="" treatment="" periods="" (5="" days="" each)="" that="" were="" separated="" by="" a="" 2-day="" washout="" period="" that="" included="" a="" 2-day="" drug-free="" period="" after="" the="" last="" treatment="" period.="" thus,="" each="" subject="" received="" each="" of="" the="" four="" treatments="" (quinine="" sulfate="" 64.8="" mg="" in="" combination="" with="" 400="" i.u.="" vitamin="" e,="" 64.8="" mg="" quinine="" sulfate,="" 400="" i.u.="" vitamin="" e,="" and="" placebo).="" a="" total="" of="" 205="" subjects="" (out="" of="" 209="" subjects="" originally="" enrolled)="" completed="" the="" study="" at="" the="" two="" locations.="" each="" morning="" upon="" arising,="" subjects="" recorded="" on="" a="" daily="" evaluation="" form="" their="" response="" to="" questions="" regarding="" their="" difficulty="" or="" failure="" to="" get="" to="" sleep="" due="" to="" night="" leg="" cramps="" and="" whether="" or="" not="" the="" cramps="" had="" awakened="" them="" the="" previous="" night.="" subjects="" were="" also="" asked="" to="" rate="" on="" a="" scale="" from="" 0="" (no="" cramps)="" to="" 3="" (very="" difficult)="" the="" effect="" of="" leg="" cramps="" on="" their="" ability="" to="" fall="" asleep="" and="" to="" record="" the="" number,="" time="" of="" occurrence,="" duration,="" and="" severity="" of="" leg="" cramps="" on="" the="" evaluation="" form.="" at="" the="" end="" of="" each="" weekly="" treatment="" period,="" subjects="" were="" asked="" to="" complete="" a="" global="" evaluation="" form="" and="" to="" record="" any="" change="" in="" their="" condition="" during="" that="" period,="" as="" follows:="" greatly="" improved,="" slightly="" improved,="" no="" improvement,="" or="" worse.="" subjects="" who="" selected="" ``worse''="" were="" asked="" to="" explain="" why.="" the="" comment's="" statistical="" analysis="" of="" the="" study="" evaluated="" the="" following="" variables="" based="" on="" portions="" of="" the="" subjects'="" daily="" evaluation="" forms="" and="" their="" global="" evaluation="" of="" treatment="" effect:="" (1)="" number="" of="" nights="" per="" week="" subjects="" had="" difficulty="" getting="" to="" sleep="" due="" to="" night="" leg="" cramps,="" (2)="" effect="" of="" leg="" cramps="" on="" subject's="" ability="" to="" get="" to="" sleep,="" (3)="" number="" of="" nights="" per="" week="" that="" leg="" cramps="" prevented="" subjects="" from="" going="" to="" sleep,="" (4)="" number="" of="" nights="" per="" week="" that="" leg="" cramps="" woke="" subjects="" up,="" (5)="" number="" of="" leg="" cramps="" per="" week,="" (6)="" severity="" of="" the="" leg="" cramps,="" and="" (7)="" subjects'="" global="" evaluations="" of="" how="" their="" condition="" changed="" over="" the="" previous="" week.="" in="" addition,="" the="" following="" parameters="" were="" derived="" from="" these="" variables="" and="" evaluated:="" (1)="" number="" of="" nights="" per="" week="" with="" leg="" cramps,="" (2)="" mean="" number="" of="" leg="" cramps="" per="" night,="" (3)="" total="" severity="" score="" during="" each="" week,="" (4)="" mean="" effect="" of="" leg="" cramps="" on="" sleep="" per="" week,="" and="" (5)="" mean="" severity="" per="" cramp.="" separate="" analyses="" of="" the="" results="" from="" each="" site="" and="" analysis="" of="" pooled="" results="" from="" both="" study="" cites="" were="" reported.="" vitamin="" e="" was="" found="" to="" be="" statistically="" significantly="" superior="" to="" placebo="" in="" 7="" of="" the="" 12="" efficacy="" variables="" evaluated="" on="" the="" basis="" of="" the="" combined="" data="" and="" in="" 6="" of="" the="" 12="" variables="" on="" the="" basis="" of="" data="" from="" at="" least="" one="" of="" the="" locations.="" the="" combination="" was="" found="" to="" be="" statistically="" superior="" to="" the="" individual="" ingredients="" and="" placebo="" on="" 11="" out="" of="" the="" 12="" variables="" evaluated="" on="" the="" basis="" of="" both="" the="" combined="" data="" and="" data="" from="" at="" least="" one="" of="" the="" locations.="" on="" that="" same="" basis,="" quinine="" sulfate="" was="" found="" to="" be="" statistically="" superior="" to="" placebo="" in="" 9="" of="" the="" 12="" variables="" evaluated="" and="" to="" vitamin="" e="" in="" 1="" of="" the="" 12="" variables.="" the="" comment="" concluded="" that="" quinine="" and="" vitamin="" e="" were="" significantly="" additive="" in="" their="" effects,="" and="" that="" it="" was="" this="" additive="" effect="" that="" resulted="" in="" the="" highly="" significant="" superiority="" of="" the="" combination="" over="" its="" individual="" components.="" the="" agency="" has="" determined="" that="" the="" statistical="" analysis="" presented="" with="" this="" study="" is="" inadequate="" for="" review="" because="" the="" model="" used="" does="" not="" properly="" separate="" the="" carryover="" effect="" from="" the="" treatment="" effect.="" the="" model="" consisted="" of="" a="" sequence="" or="" code="" effect,="" a="" subject="" within="" code="" effect,="" a="" visit="" effect,="" and="" a="" treatment="" effect.="" for="" a="" given="" subject,="" this="" model="" says="" that="" code="" effect="" is="" constant="" over="" all="" visits;="" thus,="" carryover="" effect="" must="" be="" partially="" confounded="" with="" treatment="" effect.="" therefore,="" the="" analysis="" presented="" cannot="" be="" relied="" upon="" to="" demonstrate="" the="" efficacy="" of="" any="" of="" the="" treatments.="" the="" third="" clinical="" study="" was="" a="" multicenter,="" randomized,="" block,="" parallel-design="" with="" a="" single-blind,="" placebo,="" run-in="" period,="" followed="" by="" a="" 2-week="" double-blind,="" randomized,="" treatment="" phase="" (see="" section="" i.c.,="" comment="" 12).="" no="" statistically="" significant="" treatment="" effect="" of="" vitamin="" e="" was="" detected="" at="" the="" end="" of="" the="" double-blind="" phase="" for="" any="" variable="" in="" this="" study.="" the="" agency="" concludes="" that="" the="" submitted="" data="" are="" inadequate="" to="" establish="" the="" effectiveness="" of="" vitamin="" e="" or="" the="" combination="" of="" vitamin="" e="" and="" quinine="" sulfate="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" therefore,="" both="" vitamin="" e="" individually="" and="" in="" combination="" with="" quinine="" sulfate="" are="" nonmonograph="" conditions.="" the="" agency's="" detailed="" comments="" and="" evaluation="" of="" the="" data="" are="" on="" file="" in="" the="" dockets="" management="" branch="" (refs.="" 4="" and="" 5).="" references="" (1)="" biodesign="" gmbh,="" ``clinical="" evaluation="" of="" q-velr="" in="" patients="" with="" nocturnal="" leg="" muscle="" cramps,''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" sup00031,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (2)="" leo="" winter="" associates,="" inc.,="" ``final="" medical="" report="" and="" data="" summary="" analysis="" and="" final="" statistical="" report="" on="" double="" blind="" randomized="" crossover="" study="" of="" q-velr="" versus="" quinine="" sulfate="" versus="" vitamine="" e="" versus="" placebo="" in="" the="" treatment="" of="" nocturnal="" leg="" muscle="" cramps="" (no.="" 1285-5082),''="" draft="" of="" an="" unpublished="" paper="" in="" comment="" no.="" sup00031,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (3)="" draft="" of="" an="" unpublished="" study="" entitled="" ``a="" short-term,="" randomized,="" double-blind,="" parallel="" study="" of="" q-vel="" vs.="" quinine="" sulfate="" vs.="" vitamin="" e="" vs.="" placebo="" in="" the="" prevention="" and="" treatment="" of="" nocturnal="" leg="" cramps,''="" comment="" no.="" sup00033,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (4)="" letter="" from="" w.="" e.="" gilbertson,="" fda,="" to="" l.="" d.="" fantasia,="" ciba="" consumer="" pharmaceuticals,="" coded="" let00060,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" (5)="" memorandum="" of="" telephone="" conversation="" between="" l.="" fantasia,="" ciba="" consumer="" pharmaceuticals,="" and="" l.="" geismar,="" fda,="" january="" 4,="" 1989,="" coded="" mt00009,="" docket="" no.="" 77n-0094,="" dockets="" management="" branch.="" d.="" comments="" on="" labeling="" 14.="" several="" comments="" requested="" revisions="" in="" parts="" of="" the="" labeling="" proposed="" in="" the="" tentative="" final="" monograph.="" two="" comments="" disagreed="" with="" the="" agency's="" statement="" of="" identity.="" one="" comment="" argued="" that="" it="" was="" a="" restatement="" of="" the="" indication="" proposed="" in="" sec.="" 343.150(b).="" in="" place="" of="" the="" agency's="" proposed="" statement="" of="" identity="" (``nocturnal="" leg="" muscle="" cramps="" treatment="" and/or="" prevention''),="" one="" comment="" requested="" that="" ``muscle="" relaxant="" pain="" reliever''="" or="" ``analgesic''="" be="" used.="" the="" comment="" contended="" that="" its="" suggestions="" were="" more="" descriptive="" of="" general="" pharmacological="" categories="" as="" described="" in="" 21="" cfr="" 201.61.="" another="" comment="" suggested="" changing="" the="" statement="" to="" ``night="" leg="" cramp="" relief,''="" arguing="" that="" this="" statement="" would="" be="" more="" ``meaningful="" to="" the="" layman''="" in="" accord="" with="" 21="" cfr="" 201.61.="" the="" comment="" added="" that="" its="" suggested="" term="" is="" currently="" used="" in="" the="" labeling="" of="" a="" major="" otc="" quinine="" product="" and="" reflects="" a="" more="" contemporary="" description="" of="" the="" condition="" being="" treated.="" referring="" to="" the="" warning="" proposed="" in="" sec.="" 343.150(c)="" that="" reads="" ``discontinue="" use="" if="" ringing="" in="" the="" ears,="" deafness,="" skin="" rash,="" or="" visual="" disturbances="" occur,''="" one="" comment="" requested="" that="" the="" words="" ``and="" consult="" a="" physician''="" be="" added="" following="" ``discontinue="" use.''="" the="" comment="" believed="" that="" such="" a="" warning="" would="" facilitate="" further="" medical="" treatment,="" if="" deemed="" necessary.="" the="" comment="" added="" that="" the="" agency="" had="" proposed="" similar="" warnings="" in="" other="" otc="" drug="" monographs,="" for="" example,="" proposed="" sec.="" 333.50(c)(2)="" and="" (c)(3)="" for="" topical="" acne="" drug="" products="" (january="" 15,="" 1985,="" 50="" fr="" 2172="" at="" 2181).="" the="" comment="" explained="" that="" this="" addition="" to="" the="" warning="" would="" better="" serve="" the="" elderly,="" the="" population="" most="" likely="" to="" use="" the="" product.="" one="" comment="" recommended="" that="" the="" agency="" distinguish="" between="" treatment="" and="" prevention="" directions="" for="" the="" drug,="" and="" proposed="" the="" following:="" ``when="" night="" leg="" cramps="" occur,="" take="" 200-325="" mg="" at="" once.="" to="" help="" prevent="" further="" night="" leg="" cramps,="" take="" 200-325="" mg="" two="" hours="" before="" bedtime="" for="" 14="" days.="" do="" not="" exceed="" more="" than="" 325="" mg="" daily.''="" the="" comment="" concluded="" that,="" in="" providing="" adequate="" directions="" for="" use,="" it="" is="" appropriate="" to="" discuss="" dosages="" for="" initial="" onset="" of="" leg="" muscle="" cramps="" and="" for="" prevention="" of="" future="" cramps.="" no="" ingredients="" for="" treating="" and/or="" preventing="" nocturnal="" leg="" muscle="" cramps="" are="" currently="" generally="" recognized="" as="" safe="" and="" effective="" for="" inclusion="" in="" an="" otc="" drug="" monograph;="" thus,="" no="" otc="" labeling="" is="" being="" finalized="" at="" this="" time.="" accordingly,="" the="" comments'="" requests="" are="" not="" being="" addressed="" in="" this="" document.="" however,="" in="" the="" event="" that="" any="" ingredient="" for="" treating="" and/or="" preventing="" nocturnal="" leg="" muscle="" cramps="" reaches="" otc="" drug="" monograph="" status,="" the="" agency="" will="" determine="" appropriate="" labeling="" at="" that="" time="" and="" publish="" it="" in="" a="" future="" issue="" of="" the="" federal="" register.="" ii.="" the="" agency's="" final="" conclusions="" on="" otc="" drug="" products="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" the="" agency="" concludes="" that="" the="" data="" and="" information="" submitted="" are="" inadequate="" to="" establish="" the="" safety="" and="" effectiveness="" of="" quinine="" sulfate,="" vitamin="" e,="" or="" the="" combination="" of="" quinine="" sulfate="" and="" vitamin="" e="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" three="" clinical="" studies="" of="" vitamin="" e,="" alone="" or="" in="" combination="" with="" quinine="" sulfate,="" were="" submitted.="" the="" report="" of="" one="" of="" the="" studies="" provided="" no="" details="" of="" the="" statistical="" analysis="" conducted;="" the="" model="" described="" in="" the="" summary="" of="" the="" analysis="" failed="" to="" separate="" carryover="" effect="" from="" treatment="" effect;="" and="" neither="" the="" protocol="" nor="" the="" individual="" subject="" data="" were="" provided.="" independent="" verification="" of="" the="" conclusions="" presented,="" therefore,="" was="" not="" possible.="" on="" the="" basis="" of="" the="" information="" provided="" in="" the="" report,="" no="" conclusions="" about="" the="" efficacy="" of="" vitamin="" e="" are="" possible="" from="" this="" study.="" in="" another="" study,="" a="" statistically="" significant="" effect="" of="" vitamin="" e="" was="" reported="" in="" 7="" of="" 12="" endpoints,="" and="" statistically="" significant="" differences="" from="" placebo="" were="" reported="" in="" 11="" of="" 12="" endpoints="" for="" the="" combination="" product.="" in="" this="" study,="" however,="" treatment="" effect="" was="" confounded="" by="" carryover="" effect="" making="" it="" impossible="" to="" ascribe="" observed="" differences="" to="" vitamin="" e.="" further,="" the="" third="" study,="" a="" large,="" multicenter,="" 2-week,="" parallel-design="" study="" comparing="" vitamin="" e,="" quinine="" sulfate,="" a="" combination="" of="" vitamin="" e="" and="" quinine="" sulfate,="" and="" placebo="" showed="" no="" significant="" difference="" for="" vitamin="" e="" compared="" to="" placebo="" on="" any="" parameter="" at="" the="" end="" of="" the="" double-blind="" treatment="" period.="" six="" clinical="" trials="" were="" submitted="" to="" establish="" the="" safety="" and="" efficacy="" of="" quinine="" sulfate="" in="" treating="" and/or="" preventing="" nocturnal="" leg="" muscle="" cramps.="" effectiveness="" results="" reported="" as="" significant="" were="" not="" replicated="" within="" or="" between="" studies.="" in="" two="" crossover="" studies,="" significant="" differences="" between="" quinine="" sulfate="" and="" placebo="" were="" seen="" only="" in="" the="" second="" leg="" of="" the="" crossover,="" and="" there="" were="" significant="" pretreatment="" differences.="" analysis="" of="" the="" first="" leg="" of="" these="" crossover="" studies="" showed="" no="" effect="" of="" quinine.="" in="" a="" large,="" 2-week,="" parallel="" study="" of="" quinine="" sulfate,="" vitamin="" e,="" and="" the="" combination="" of="" these="" ingredients="" versus="" placebo,="" no="" statistically="" significant="" differences="" were="" found="" between="" active="" treatments="" and="" placebo="" for="" the="" full="" 2="" weeks="" of="" the="" study.="" furthermore,="" each="" study="" involved="" multiple="" endpoints,="" none="" of="" which="" was="" prospectively="" declared="" as="" the="" primary="" efficacy="" variable(s)="" in="" any="" study.="" statistical="" analysis="" was="" conducted="" without="" regard="" to="" adjustment="" for="" multiple="" comparisons,="" casting="" doubt="" on="" the="" validity="" of="" claimed="" statistical="" significance="" in="" many="" cases.="" in="" three="" crossover="" studies,="" the="" treatment="" effect="" was="" confounded="" by="" potential="" carryover="" effect="" making="" it="" impossible="" to="" attribute="" the="" results="" to="" the="" study="" drugs.="" the="" agency="" concludes="" that="" the="" data="" and="" information="" submitted="" do="" not="" provide="" substantial="" evidence="" of="" effectiveness="" of="" quinine="" sulfate,="" vitamin="" e,="" or="" a="" combination="" of="" quinine="" sulfate="" and="" vitamin="" e,="" in="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" finally,="" new="" information="" has="" raised="" serious="" safety="" concerns="" over="" the="" otc="" availability="" of="" quinine="" sulfate="" for="" this="" use.="" adverse="" events="" characteristic="" of="" quinine="" toxicity="" were="" observed="" in="" the="" healthy="" populations="" enrolled="" in="" the="" clinical="" efficacy="" studies="" at="" doses="" of="" 260="" mg="" and="" 325="" mg="" daily.="" these="" events="" included:="" visual,="" auditory,="" and="" gastrointestinal="" symptoms,="" and="" fever.="" studies="" of="" auditory,="" vestibular,="" and="" visual="" function="" in="" subjects="" given="" quinine="" confirm="" sensory="" disturbances="" at="" even="" lower="" doses.="" altered="" pharmacokinetics="" with="" age="" results="" in="" a="" longer="" half-life="" of="" quinine="" in="" older="" people="" that="" suggests="" the="" frequency="" and="" severity="" of="" adverse="" effects="" may="" be="" greater="" in="" the="" elderly.="" in="" addition="" to="" these="" adverse="" effects,="" serious="" and="" unpredictable="" hypersensitivity="" reactions="" to="" quinine="" occur.="" symptoms="" are="" often="" dramatic,="" leading="" people="" to="" seek="" medical="" treatment.="" hospitalization="" may="" be="" required,="" and="" fatalities="" have="" been="" reported.="" while="" quinine-induced="" thrombocytopenia="" is="" the="" hypersensitivity="" reaction="" most="" frequently="" reported="" to="" the="" agency's="" spontaneous="" reporting="" system,="" estimates="" of="" the="" incidence="" of="" quinine-induced="" thrombocytopenia="" are="" unreliable.="" estimates="" based="" on="" the="" most="" direct="" evidence,="" however,="" suggest="" occurrence="" rates="" between="" 1:1,000="" and="" 1:3,500.="" quinine="" is="" the="" only="" drug="" available="" otc="" that="" has="" such="" a="" high="" association="" with="" this="" serious="" hematologic="" sensitivity.="" because="" there="" are="" no="" known="" factors="" that="" predispose="" people="" to="" the="" development="" of="" hypersensitivity="" to="" quinine,="" which="" may="" occur="" after="" 1="" week="" of="" exposure="" or="" after="" months="" or="" years="" of="" use,="" label="" warnings="" cannot="" be="" expected="" to="" protect="" consumers="" from="" hypersensitivity="" reactions="" to="" quinine="" products.="" given="" the="" benign="" nature="" of="" nocturnal="" leg="" muscle="" cramps,="" the="" failure="" of="" the="" clinical="" studies="" to="" demonstrate="" efficacy="" of="" quinine="" sulfate="" in="" this="" condition,="" the="" evidence="" of="" symptoms="" of="" quinine="" toxicity="" at="" the="" otc="" doses="" employed="" for="" leg="" cramps="" in="" a="" proportion="" of="" the="" target="" population,="" and="" the="" potential="" for="" serious,="" life="" threatening,="" and="" fatal="" hypersensitivity="" reactions="" to="" quinine,="" the="" agency="" concludes="" that="" quinine="" is="" not="" safe="" for="" otc="" use="" in="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" no="" comments="" were="" received="" in="" response="" to="" the="" agency's="" request="" for="" specific="" comment="" on="" the="" economic="" impact="" of="" this="" rulemaking="" (47="" fr="" 43562="" and="" 50="" fr="" 46588="" at="" 46593).="" an="" analysis="" of="" the="" cost="" and="" benefits="" of="" this="" regulatuion,="" conducted="" under="" executive="" order="" 12291,="" was="" discussed="" in="" the="" tentative="" final="" rule="" of="" november="" 8,="" 1985,="" (50="" fr="" 46588).="" no="" comments="" were="" received="" in="" response="" to="" the="" agencies="" tentative="" final="" rule,="" and="" the="" substances="" of="" that="" analysis="" has="" not="" changed.="" executive="" order="" 12291="" has="" been="" superseded="" by="" executive="" order="" 12866.="" fda="" has="" examined="" the="" impacts="" of="" the="" final="" rule="" under="" executive="" order="" 12866="" and="" the="" regulatory="" flexibility="" act="" (pub.="" l.="" 96-354).="" executive="" order="" 12866="" directs="" agencies="" to="" assess="" all="" costs="" and="" benefits="" of="" available="" regulatory="" alternatives="" and,="" when="" regulation="" is="" necessary,="" to="" select="" regulatory="" approaches="" that="" maximize="" net="" benefits="" (including="" potential="" economic,="" environmental,="" public="" health="" and="" safety,="" and="" other="" advantages;="" distributive="" impacts;="" and="" equity).="" the="" agency="" believes="" that="" this="" final="" rule="" is="" consistent="" with="" the="" regulatory="" philosophy="" and="" principles="" identified="" in="" the="" executive="" order.="" in="" addition,="" the="" final="" rule="" is="" not="" a="" significant="" regulatory="" action="" as="" defined="" by="" the="" executive="" order="" and,="" thus,="" is="" not="" subject="" to="" review="" under="" the="" executive="" order.="" the="" regulatory="" flexibility="" act="" requires="" agencies="" to="" analyze="" regulatory="" options="" that="" would="" minimize="" any="" significant="" impact="" of="" a="" rule="" on="" small="" entities.="" although="" the="" final="" rule="" will="" result="" in="" the="" removal="" of="" some="" products="" from="" the="" otc="" marketplace,="" only="" a="" limited="" number="" of="" products="" are="" affected.="" these="" include:="" (1)="" all="" combination="" products="" containing="" quinine="" sulfate="" and="" vitamin="" e,="" (2)="" products="" containing="" quinine="" sulfate="" alone="" labeled="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps,="" (3)="" products="" containing="" vitamin="" e="" alone="" labeled="" with="" the="" same="" claim,="" and="" (4)="" any="" other="" products="" marketed="" otc="" for="" this="" claim.="" no="" further="" initial="" introduction="" or="" delivery="" for="" introduction="" into="" interstate="" commerce="" of="" any="" otc="" drug="" product="" labeled="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" will="" be="" allowed="" after="" the="" effective="" date="" of="" this="" final="" rule.="" quinine="" is="" currently="" available="" as="" an="" otc="" drug="" for="" treating="" chills="" and="" fever="" of="" malaria.="" based="" on="" an="" agency="" review="" of="" currently="" marketed="" products,="" it="" appears="" that="" approximately="" two-thirds="" of="" these="" quinine-containing="" products="" are="" marketed="" for="" antimalarial="" use="" (with="" approximately="" one-="" third="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps).="" (otc="" quinine="" drug="" products="" for="" antimalarial="" use="" will="" be="" discussed="" in="" future="" issues="" of="" the="" federal="" register.)="" vitamin="" e="" is="" currently="" available="" otc="" for="" use="" as="" a="" vitamin.="" this="" final="" rule="" does="" not="" affect="" the="" continued="" marketing="" and="" availability="" of="" products="" containing="" this="" vitamin="" provided="" the="" products="" are="" not="" labeled="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" products="" containing="" quinine="" sulfate="" and/or="" vitamin="" e="" may="" be="" relabeled="" and="" reformulated="" where="" necessary="" (e.g.,="" combination="" products)="" and="" remain="" in="" the="" marketplace="" with="" other="" allowed="" claims,="" as="" described="" above.="" accordingly,="" the="" agency="" certifies="" that="" the="" final="" rule="" will="" not="" have="" a="" significant="" economic="" impact="" on="" a="" substantial="" number="" of="" small="" entities.="" therefore,="" under="" the="" regulatory="" flexibility="" act,="" no="" further="" analysis="" is="" required.="" the="" agency="" has="" determined="" under="" 21="" cfr="" 25.24(c)(6)="" that="" this="" action="" is="" of="" a="" type="" that="" does="" not="" individually="" or="" cumulatively="" have="" a="" significant="" effect="" on="" the="" human="" environment.="" therefore,="" neither="" an="" environmental="" assessment="" nor="" an="" environmental="" impact="" statement="" is="" required.="" list="" of="" subjects="" in="" 21="" cfr="" part="" 310="" administrative="" practice="" and="" procedure,="" drugs,="" labeling,="" medical="" devices,="" reporting="" and="" recordkeeping="" requirements.="" therefore,="" under="" the="" federal="" food,="" drug,="" and="" cosmetic="" act="" and="" under="" authority="" delegated="" to="" the="" commissioner="" of="" food="" and="" drugs,="" 21="" cfr="" part="" 310="" is="" amended="" as="" follows:="" part="" 310--new="" drugs="" 1.="" the="" authority="" citation="" for="" 21="" cfr="" part="" 310="" continues="" to="" read="" as="" follows:="" authority:="" secs.="" 201,="" 301,="" 501,="" 502,="" 503,="" 505,="" 506,="" 507,="" 512-="" 516,="" 520,="" 601(a),="" 701,="" 704,="" 705,="" 721="" of="" the="" federal="" food,="" drug,="" and="" cosmetic="" act="" (21="" u.s.c.="" 321,="" 331,="" 351,="" 352,="" 353,="" 355,="" 356,="" 357,="" 360b-360f,="" 360j,="" 361(a),="" 371,="" 374,="" 375,="" 379e);="" secs.="" 215,="" 301,="" 302(a),="" 351,="" 354-360f="" of="" the="" public="" health="" service="" act="" (42="" u.s.c.="" 216,="" 241,="" 242(a),="" 262,="" 263b-263n).="" 2.="" new="" sec.="" 310.546="" is="" added="" to="" subpart="" e="" to="" read="" as="" follows:="" sec.="" 310.546="" drug="" products="" containing="" active="" ingredients="" offered="" over-="" the-counter="" (otc)="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" (a)="" quinine="" sulfate="" alone="" or="" in="" combination="" with="" vitamin="" e="" has="" been="" present="" in="" over-the-counter="" (otc)="" drug="" products="" for="" the="" treatment="" and/="" or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps,="" i.e.,="" a="" condition="" of="" localized="" pain="" in="" the="" lower="" extremities="" usually="" occurring="" in="" middle="" life="" and="" beyond="" with="" no="" regular="" pattern="" concerning="" time="" or="" severity.="" there="" is="" a="" lack="" of="" adequate="" data="" to="" establish="" general="" recognition="" of="" the="" safety="" and="" effectiveness="" of="" quinine="" sulfate,="" vitamin="" e,="" or="" any="" other="" ingredients="" for="" otc="" use="" in="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" in="" the="" doses="" used="" to="" treat="" or="" prevent="" this="" condition,="" quinine="" sulfate="" has="" caused="" adverse="" events="" such="" as="" transient="" visual="" and="" auditory="" disturbances,="" dizziness,="" fever,="" nausea,="" vomiting,="" and="" diarrhea.="" quinine="" sulfate="" may="" cause="" unpredictable="" serious="" and="" life-="" threatening="" hypersensitivity="" reactions="" requiring="" medical="" intervention="" and="" hospitalization;="" fatalities="" have="" been="" reported.="" the="" risk="" associated="" with="" use="" of="" quinine="" sulfate,="" in="" the="" absence="" of="" evidence="" of="" its="" effectiveness,="" outweighs="" any="" potential="" benefit="" in="" treating="" and/or="" preventing="" this="" benign,="" self-limiting="" condition.="" based="" upon="" the="" adverse="" benefit-to-risk="" ratio,="" any="" drug="" product="" containing="" quinine="" or="" quinine="" sulfate="" cannot="" be="" considered="" generally="" recognized="" as="" safe="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps.="" (b)="" any="" otc="" drug="" product="" that="" is="" labeled,="" represented,="" or="" promoted="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" is="" regarded="" as="" a="" new="" drug="" within="" the="" meaning="" of="" section="" 201(p)="" of="" the="" federal="" food,="" drug,="" and="" cosmetic="" act="" (the="" act),="" for="" which="" an="" approved="" application="" or="" abbreviated="" application="" under="" section="" 505="" of="" the="" act="" and="" part="" 314="" of="" this="" chapter="" is="" required="" for="" marketing.="" in="" the="" absence="" of="" an="" approved="" new="" drug="" application="" or="" abbreviated="" new="" drug="" application,="" such="" product="" is="" also="" misbranded="" under="" section="" 502="" of="" the="" act.="" (c)="" clinical="" investigations="" designed="" to="" obtain="" evidence="" that="" any="" drug="" product="" labeled,="" represented,="" or="" promoted="" for="" otc="" use="" for="" the="" treatment="" and/or="" prevention="" of="" nocturnal="" leg="" muscle="" cramps="" is="" safe="" and="" effective="" for="" the="" purpose="" intended="" must="" comply="" with="" the="" requirements="" and="" procedures="" governing="" the="" use="" of="" investigational="" new="" drugs="" set="" forth="" in="" part="" 312="" of="" this="" chapter.="" (d)="" after="" february="" 22,="" 1995,="" any="" such="" otc="" drug="" product="" initially="" introduced="" or="" initially="" delivered="" for="" introduction="" into="" interstate="" commerce="" that="" is="" not="" in="" compliance="" with="" this="" section="" is="" subject="" to="" regulatory="" action.="" dated:="" august="" 4,="" 1994.="" michael="" r.="" taylor,="" deputy="" commissioner="" for="" policy.="" [fr="" doc.="" 94-20449="" filed="" 8-19-94;="" 8:45="" am]="" billing="" code="" 4160-01-f="">

Document Information

Published:
08/22/1994
Entry Type:
Uncategorized Document
Action:
Final rule.
Document Number:
94-20449
Dates:
February 22, 1995.
Pages:
0-0 (1 pages)
Docket Numbers:
Federal Register: August 22, 1994
CFR: (2)
21 CFR 330.10(a)(4)(ii)
21 CFR 310.546